相关参考文献
注意：仅列出部分参考文献，下载原文获取全部文献信息。Telmisartan and thiorphan combination treatment attenuates fibrosis and apoptosis in preventing diabetic cardiomyopathy
Vajir Malek et al.
CARDIOVASCULAR RESEARCH (2019)
Inhibition of JNK and p38 MAPK-mediated inflammation and apoptosis by ivabradine improves cardiac function in streptozotocin-induced diabetic cardiomyopathy
Guangfeng Zuo et al.
JOURNAL OF CELLULAR PHYSIOLOGY (2019)
LCZ696 (Sacubitril/Valsartan), an Angiotensin-Receptor Neprilysin Inhibitor, Attenuates Cardiac Hypertrophy, Fibrosis, and Vasculopathy in a Rat Model of Chronic Kidney Disease
Yasunori Suematsu et al.
JOURNAL OF CARDIAC FAILURE (2018)
Impact of sacubitril-valsartan combination in patients with chronic heart failure and sleep apnoea syndrome: the ENTRESTO-SAS study design
Dany Jaffuel et al.
ESC HEART FAILURE (2018)
Hyperglycemia- and hyperinsulinemia-induced insulin resistance causes alterations in cellular bioenergetics and activation of inflammatory signaling in lymphatic muscle
Yang Lee et al.
FASEB JOURNAL (2017)
LCZ696, an angiotensin receptor-neprilysin inhibitor, improves cardiac function with the attenuation of fibrosis in heart failure with reduced ejection fraction in streptozotocin-induced diabetic mice
Yasunori Suematsu et al.
EUROPEAN JOURNAL OF HEART FAILURE (2016)
Atorvastatin Alleviates Experimental Diabetic Cardiomyopathy by Regulating the GSK-3β-PP2Ac-NF-κB Signaling Axis
Xiao-min Ree et al.
PLOS ONE (2016)
Genistein alleviates pressure overload-induced cardiac dysfunction and interstitial fibrosis in mice
Wei Qin et al.
BRITISH JOURNAL OF PHARMACOLOGY (2015)
LCZ696 (Sacubitril/Valsartan) Can We Predict Who Will Benefit?
G. William Dec
JOURNAL OF THE AMERICAN COLLEGE OF CARDIOLOGY (2015)
Inhibition of JNK Phosphorylation by a Novel Curcumin Analog Prevents High Glucose-Induced Inflammation and Apoptosis in Cardiomyocytes and the Development of Diabetic Cardiomyopathy
Yong Pan et al.
DIABETES (2014)
The potential role of valsartan+AHU377 (LCZ696) in the treatment of heart failure
Adriaan A. Voors et al.
EXPERT OPINION ON INVESTIGATIONAL DRUGS (2013)
Retinoic acid protects cardiomyocytes from high glucose-induced apoptosis through inhibition of NF-κB signaling pathway
Irina T. Nizamutdinova et al.
JOURNAL OF CELLULAR PHYSIOLOGY (2013)
NADPH oxidase-derived superoxide Anion-induced apoptosis is mediated via the JNK-dependent activation of NF-κB in cardiomyocytes exposed to high glucose
Kun-Hsi Tsai et al.
JOURNAL OF CELLULAR PHYSIOLOGY (2012)
The angiotensin receptor neprilysin inhibitor LCZ696 in heart failure with preserved ejection fraction: a phase 2 double-blind randomised controlled trial
Scott D. Solomon et al.
LANCET (2012)
Cardiomyocyte NF-kappa B p65 promotes adverse remodelling, apoptosis, and endoplasmic reticulum stress in heart failure
Tariq Hamid et al.
CARDIOVASCULAR RESEARCH (2011)
Cannabidiol protects against hepatic ischemia/reperfusion injury by attenuating inflammatory signaling and response, oxidative/nitrative stress, and cell death
Partha Mukhopadhyay et al.
FREE RADICAL BIOLOGY AND MEDICINE (2011)
National, regional, and global trends in fasting plasma glucose and diabetes prevalence since 1980: systematic analysis of health examination surveys and epidemiological studies with 370 country-years and 2.7 million participants
Goodarz Danaei et al.
LANCET (2011)
Oxidative stress: a key contributor to diabetic cardiomyopathy
Madhu Khullar et al.
CANADIAN JOURNAL OF PHYSIOLOGY AND PHARMACOLOGY (2010)
Nitric oxide mediates cardiac protection of tissue kallikrein by reducing inflammation and ventricular remodeling after myocardial ischemia/reperfusion
Hang Yin et al.
LIFE SCIENCES (2008)
Tumor necrosis factor-alpha antagonism protects from myocardial inflammation and fibrosis in experimental diabetic cardiomyopathy
Dirk Westermann et al.
BASIC RESEARCH IN CARDIOLOGY (2007)
Protection against oxidative stress in diabetic rats:: Role of angiotensin AT1 receptor and beta 1-adrenoceptor antagonism
M Dorenkamp et al.
EUROPEAN JOURNAL OF PHARMACOLOGY (2005)
c-Jun N-terminal kinases (JNK) antagonize cardiac growth through cross-talk with calcineurin-NFAT signaling
QR Liang et al.
EMBO JOURNAL (2003)
Hypertensive end-organ damage and premature mortality are p38 mitogen-activated protein kinase-dependent in a rat model of cardiac hypertrophy and dysfunction
TM Behr et al.
CIRCULATION (2001)
分享论文
