相关参考文献
注意:仅列出部分参考文献,下载原文获取全部文献信息。Inhibition of RAGE Axis Signaling: A Pharmacological Challenge
Armando Rojas et al.
CURRENT DRUG TARGETS (2019)
Targeting RAGE Signaling in Inflammatory Disease
Barry I. Hudson et al.
ANNUAL REVIEW OF MEDICINE, VOL 69 (2018)
Oxidized low-density lipoprotein promotes osteoclast differentiation from CD68 positive mononuclear cells by regulating HMGB1 release
Chunfeng Hou et al.
BIOCHEMICAL AND BIOPHYSICAL RESEARCH COMMUNICATIONS (2018)
sRAGE prolonged stem cell survival and suppressed RAGE-related inflammatory cell and T lymphocyte accumulations in an Alzheimer's disease model
Seyeon Oh et al.
BIOCHEMICAL AND BIOPHYSICAL RESEARCH COMMUNICATIONS (2018)
HMGB1-induced inflammatory response promotes bone healing in murine tooth extraction socket
Hiroaki Aoyagi et al.
JOURNAL OF CELLULAR BIOCHEMISTRY (2018)
RAGE-dependent mitochondria pathway: a novel target of silibinin against apoptosis of osteoblastic cells induced by advanced glycation end products
Y. X. Mao et al.
CELL DEATH & DISEASE (2018)
A prospective study of soluble receptor for advanced glycation end products and adipokines in association with pancreatic cancer in postmenopausal women
Donna L. White et al.
CANCER MEDICINE (2018)
Advanced Glycation End Products and esRAGE Are Associated With Bone Turnover and Incidence of Hip Fracture in Older Men
Lydia S. Lamb et al.
JOURNAL OF CLINICAL ENDOCRINOLOGY & METABOLISM (2018)
Advanced glycation end products induced IL-6 and VEGF-A production and apoptosis in osteocyte-like MLO-Y4 cells by activating RAGE and ERK1/2, P38 and STAT3 signalling pathways
Helin Chen et al.
INTERNATIONAL IMMUNOPHARMACOLOGY (2017)
Associations between Soluble Receptor for Advanced Glycation End Products (sRAGE) and S100A12 (EN-RAGE) with Mortality in Long-term Hemodialysis Patients
Eul Sik Jung et al.
JOURNAL OF KOREAN MEDICAL SCIENCE (2017)
Targeting the Receptor for Advanced Glycation Endproducts (RAGE): A Medicinal Chemistry Perspective
Salvatore Bongarzone et al.
JOURNAL OF MEDICINAL CHEMISTRY (2017)
Advanced glycation end-products produced systemically and by macrophages: A common contributor to inflammation and degenerative diseases
Kyunghee Byun et al.
PHARMACOLOGY & THERAPEUTICS (2017)
PREVALENCE AND RISK FACTORS OF OSTEOPOROSIS IN POSTMENOPAUSAL WOMEN WITH TYPE 2 DIABETES MELLITUS
Ivan Raska et al.
CENTRAL EUROPEAN JOURNAL OF PUBLIC HEALTH (2017)
Evaluation of circulating sRAGE in osteoporosis according to BMI, adipokines and fracture risk: a pilot observational study
Emanuela Galliera et al.
IMMUNITY & AGEING (2017)
Extracellular S100A4 negatively regulates osteoblast function by activating the NF-κB pathway
Haemin Kim et al.
BMB REPORTS (2017)
Autologous minced muscle grafts improve endogenous fracture healing and muscle strength after musculoskeletal trauma
Brady J. Hurtgen et al.
PHYSIOLOGICAL REPORTS (2017)
Disruption of the Cx43/miR21 pathway leads to osteocyte apoptosis and increased osteoclastogenesis with aging
Hannah M. Davis et al.
AGING CELL (2017)
Advanced Glycation End Product 3 (AGE3) Increases Apoptosis and the Expression of Sclerostin by Stimulating TGF-β Expression and Secretion in Osteocyte-Like MLO-Y4-A2 Cells
Masakazu Notsu et al.
CALCIFIED TISSUE INTERNATIONAL (2017)
The shedding-derived soluble receptor for advanced glycation endproducts sustains inflammation during acute Pseudomonas aeruginosa lung infection
Antonella Antonelli et al.
BIOCHIMICA ET BIOPHYSICA ACTA-GENERAL SUBJECTS (2017)
AGEs Induce Apoptosis in Rat Osteoblast Cells by Activating the Caspase-3 Signaling Pathway Under a High-Glucose Environment In Vitro
Jiaqiang Liu et al.
APPLIED BIOCHEMISTRY AND BIOTECHNOLOGY (2016)
The multiple faces of RAGE - opportunities for therapeutic intervention in aging and chronic disease
Ravichandran Ramasamy et al.
EXPERT OPINION ON THERAPEUTIC TARGETS (2016)
Advanced glycation end products biphasically modulate bone resorption in osteoclast-like cells
Ziqing Li et al.
AMERICAN JOURNAL OF PHYSIOLOGY-ENDOCRINOLOGY AND METABOLISM (2016)
Emerging drugs to reduce abnormal β-amyloid protein in Alzheimer's disease patients
Francesco Panza et al.
EXPERT OPINION ON EMERGING DRUGS (2016)
Regulation of Receptor for Advanced Glycation End Products (RAGE) Ectodomain Shedding and Its Role in Cell Function
Alex Braley et al.
JOURNAL OF BIOLOGICAL CHEMISTRY (2016)
RAGE-mediated inflammation in patients with septic shock
Stefan Hofer et al.
JOURNAL OF SURGICAL RESEARCH (2016)
RAGE axis in neuroinflammation, neurodegeneration and its emerging role in the pathogenesis of amyotrophic lateral sclerosis
Rashmi Ray et al.
NEUROSCIENCE AND BIOBEHAVIORAL REVIEWS (2016)
Advanced Glycation Endproducts and Bone Material Properties in Type 1 Diabetic Mice
Mishaela R. Rubin et al.
PLOS ONE (2016)
Receptor for Advanced Glycation End Products-Mediated Signaling Impairs the Maintenance of Bone Marrow Mesenchymal Stromal Cells in Diabetic Model Mice
Eriko Aikawa et al.
STEM CELLS AND DEVELOPMENT (2016)
Soluble RAGE Treatment Delays Progression of Amyotrophic Lateral Sclerosis in SOD1 Mice
Judyta K. Juranek et al.
FRONTIERS IN CELLULAR NEUROSCIENCE (2016)
HMGB1 promotes the secretion of multiple cytokines and potentiates the osteogenic differentiation of mesenchymal stem cells through the Ras/MAPK signaling pathway
Lin Feng et al.
EXPERIMENTAL AND THERAPEUTIC MEDICINE (2016)
Effects of high glucose and advanced glycation end products on the expressions of sclerostin and RANKL as well as apoptosis in osteocyte-like MLO-Y4-A2 cells
Ken-ichiro Tanaka et al.
BIOCHEMICAL AND BIOPHYSICAL RESEARCH COMMUNICATIONS (2015)
Receptor for advanced glycation endproduct modulators: a new therapeutic target in Alzheimer's disease
Douglas Walker et al.
EXPERT OPINION ON INVESTIGATIONAL DRUGS (2015)
Advanced Glycation End Products Affect Osteoblast Proliferation and Function by Modulating Autophagy Via the Receptor of Advanced Glycation End Products/Raf Protein/Mitogen-activated Protein Kinase/Extracellular Signalregulated Kinase Kinase/Extracellular Signal-regulated Kinase (RAGE/Raf/MEK/ERK) Pathway
Hong-Zheng Meng et al.
JOURNAL OF BIOLOGICAL CHEMISTRY (2015)
A direct role of collagen glycation in bone fracture
Atharva A. Poundarik et al.
JOURNAL OF THE MECHANICAL BEHAVIOR OF BIOMEDICAL MATERIALS (2015)
Effects of Curculigoside on Memory Impairment and Bone Loss via Anti-Oxidative Character in APP/PS1 Mutated Transgenic Mice
Lu Zhao et al.
PLOS ONE (2015)
Collagen advanced glycation inhibits its Discoidin Domain Receptor 2 (DDR2)-mediated induction of lysyl oxidase in osteoblasts
Roozbeh Khosravi et al.
BONE (2014)
Amyloid beta peptide is elevated in osteoporotic bone tissues and enhances osteoclast function
Shangfu Li et al.
BONE (2014)
Insights into the physiological function of the β-amyloid precursor protein: beyond Alzheimer's disease
Edgar Dawkins et al.
JOURNAL OF NEUROCHEMISTRY (2014)
Clinical trial of an inhibitor of RAGE-Aβ interactions in Alzheimer disease
Douglas Galasko et al.
NEUROLOGY (2014)
RAGE in tissue homeostasis, repair and regeneration
Guglielmo Sorci et al.
BIOCHIMICA ET BIOPHYSICA ACTA-MOLECULAR CELL RESEARCH (2013)
Intra-peritoneal sRAGE treatment induces alterations in cellular distribution of CD19+, CD3+ and Mac-1+ cells in lymphoid organs and peritoneal cavity
Mikael Brisslert et al.
CELL AND TISSUE RESEARCH (2013)
Swedish Mutant APP Suppresses Osteoblast Differentiation and Causes Osteoporotic Deficit, Which Are Ameliorated by N-Acetyl-L-Cysteine
Wen-Fang Xia et al.
JOURNAL OF BONE AND MINERAL RESEARCH (2013)
Advanced glycation end products suppress lysyl oxidase and induce bone collagen degradation in a rat model of renal osteodystrophy
Chiharu Aoki et al.
LABORATORY INVESTIGATION (2013)
Alternative Splicing of the RAGE Cytoplasmic Domain Regulates Cell Signaling and Function
Joel Jules et al.
PLOS ONE (2013)
Receptor for Advanced Glycation End Products and Its Involvement in Inflammatory Diseases
Yaw Kuang Chuah et al.
INTERNATIONAL JOURNAL OF INFLAMMATION (2013)
Is RAGE still a therapeutic target for Alzheimer's disease?
Richard J. Deane
FUTURE MEDICINAL CHEMISTRY (2012)
A multimodal RAGE-specific inhibitor reduces amyloid β-mediated brain disorder in a mouse model of Alzheimer disease
Rashid Deane et al.
JOURNAL OF CLINICAL INVESTIGATION (2012)
被撤回的出版物: Redox Modification of Cysteine Residues Regulates the Cytokine Activity of High Mobility Group Box-1 (HMGB1) (Publication with Expression of Concern. See vol. 26, 2020) (Retracted article. See vol. 26, 2020)
Huan Yang et al.
MOLECULAR MEDICINE (2012)
S100B Engages RAGE or bFGF/FGFR1 in Myoblasts Depending on Its Own Concentration and Myoblast Density. Implications for Muscle Regeneration
Francesca Riuzzi et al.
PLOS ONE (2012)
In situ accumulation of advanced glycation endproducts (AGEs) in bone matrix and its correlation with osteoclastic bone resorption
X. Neil Dong et al.
BONE (2011)
APPswe/Aβ Regulation of Osteoclast Activation and RAGE Expression in an Age-Dependent Manner
Shun Cui et al.
JOURNAL OF BONE AND MINERAL RESEARCH (2011)
Soluble Receptor for Advanced Glycation Endproducts Is Decreased in Patients with Juvenile Idiopathic Arthritis (ERA Category) and Inversely Correlates with Disease Activity and S100A12 Levels
Arpita Myles et al.
JOURNAL OF RHEUMATOLOGY (2011)
RAGE supports parathyroid hormone-induced gains in femoral trabecular bone
Binu K. Philip et al.
AMERICAN JOURNAL OF PHYSIOLOGY-ENDOCRINOLOGY AND METABOLISM (2010)
Low Serum Level of the Endogenous Secretory Receptor for Advanced Glycation End Products (esRAGE) Is a Risk Factor for Prevalent Vertebral Fractures Independent of Bone Mineral Density in Patients With Type 2 Diabetes
Masahiro Yamamoto et al.
DIABETES CARE (2009)
Alternative splicing of the murine receptor for advanced glycation end-products (RAGE) gene
Anastasia Z. Kalea et al.
FASEB JOURNAL (2009)
γ-Secretase-dependent cleavage of amyloid precursor protein regulates osteoblast behavior
Jane McLeod et al.
FASEB JOURNAL (2009)
Direct Inhibitory and Indirect Stimulatory Effects of RAGE Ligand S100 on sRANKL-Induced Osteoclastogenesis
Tomohiko Yoshida et al.
JOURNAL OF CELLULAR BIOCHEMISTRY (2009)
RAGE-mediated signaling contributes to intraneuronal transport of amyloid-β and neuronal dysfunction
Kazuhiro Takuma et al.
PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA (2009)
Soluble receptor for advanced glycation end products: a new biomarker in diagnosis and prognosis of chronic inflammatory diseases
Helene Maillard-Lefebvre et al.
RHEUMATOLOGY (2009)
The decrease of soluble RAGE levels in rheumatoid arthritis patients following hormone replacement therapy is associated with increased bone mineral density and diminished bone/cartilage turnover: a randomized controlled trial
Rille Pullerits et al.
RHEUMATOLOGY (2009)
Identification, classification, and expression of RAGE gene splice variants
Barry I. Hudson et al.
FASEB JOURNAL (2008)
Interaction of the RAGE Cytoplasmic Domain with Diaphanous-1 Is Required for Ligand-stimulated Cellular Migration through Activation of Rac1 and Cdc42
Barry I. Hudson et al.
JOURNAL OF BIOLOGICAL CHEMISTRY (2008)
HMGB1 regulates RANKL-induced osteoclastogenesis in a manner dependent on RAGE
Zheng Zhou et al.
JOURNAL OF BONE AND MINERAL RESEARCH (2008)
Is HMGB1 an osteocyte alarmin?
Joseph P. Bidwell et al.
JOURNAL OF CELLULAR BIOCHEMISTRY (2008)
HMGB1 is a bone-active cytokine
Jieping Yang et al.
JOURNAL OF CELLULAR PHYSIOLOGY (2008)
Structural and functional insights into RAGE activation by multimeric S100B
Thorsten Ostendorp et al.
EMBO JOURNAL (2007)
Advanced glycation end products stimulate osteoblast apoptosis via the MAP kinase and cytosolic apoptotic pathways
Mani Alikhani et al.
BONE (2007)
Inhibition of the RAGE products increases survival in experimental models of severe sepsis and systemic infection
Emily C. Lutterloh et al.
CRITICAL CARE (2007)
HMGB1 expression and release by bone cells
K Charoonpatrapong et al.
JOURNAL OF CELLULAR PHYSIOLOGY (2006)
Regulation of osteoclast function and bone mass by RAGE
Z Zhou et al.
JOURNAL OF EXPERIMENTAL MEDICINE (2006)
High mobility group box 1 protein interacts with multiple Toll-like receptors
JS Park et al.
AMERICAN JOURNAL OF PHYSIOLOGY-CELL PHYSIOLOGY (2006)
Disordered osteoclast formation in RAGE-deficient mouse establishes an essential role for RAGE in diabetes related bone loss
KH Ding et al.
BIOCHEMICAL AND BIOPHYSICAL RESEARCH COMMUNICATIONS (2006)
Understanding RAGE, the receptor for advanced glycation end products
A Bierhaus et al.
JOURNAL OF MOLECULAR MEDICINE-JMM (2005)
Advanced glycation end-products attenuate human mesenchymal stem cells and prevent cognate differentiation into adipose tissue, cartilage, and bone
S Kume et al.
JOURNAL OF BONE AND MINERAL RESEARCH (2005)
Extracellular HMGB1, a signal of tissue damage, induces mesoangioblast migration and proliferation
R Palumbo et al.
JOURNAL OF CELL BIOLOGY (2004)
Purification and characterization of mouse soluble receptor for advanced glycation end products (sRAGE)
LE Hanford et al.
JOURNAL OF BIOLOGICAL CHEMISTRY (2004)
S100 proteins in mouse and man: from evolution to function and pathology (including an update of the nomenclature)
I Marenholz et al.
BIOCHEMICAL AND BIOPHYSICAL RESEARCH COMMUNICATIONS (2004)
Advanced glycation end-products (AGEs) induce concerted changes in the osteoblastic expression of their receptor RAGE and in the activation of extracellular signal-regulated kinases (ERK)
AM Cortizo et al.
MOLECULAR AND CELLULAR BIOCHEMISTRY (2003)
A role for advanced glycation end products in diminished bone healing in type 1 diabetes
RB Santana et al.
DIABETES (2003)
Release of chromatin protein HMGB1 by necrotic cells triggers inflammation
P Scaffidi et al.
NATURE (2002)
Non-enzymatic glycosylation of a type I collagen matrix: effects on osteoblastic development and oxidative stress
AD McCarthy et al.
BMC CELL BIOLOGY (2001)
The biology of the receptor for advanced glycation end products and its ligands
AM Schmidt et al.
BIOCHIMICA ET BIOPHYSICA ACTA-MOLECULAR CELL RESEARCH (2000)
Cutting edge: HMG-1 as a mediator of acute lung inflammation
E Abraham et al.
JOURNAL OF IMMUNOLOGY (2000)
Blockade of RAGE suppresses periodontitis-associated bone loss in diabetic mice
E Lalla et al.
JOURNAL OF CLINICAL INVESTIGATION (2000)