期刊
JOURNAL OF ALLERGY AND CLINICAL IMMUNOLOGY
卷 142, 期 5, 页码 1415-1422出版社
MOSBY-ELSEVIER
DOI: 10.1016/j.jaci.2018.09.011
关键词
Allergic asthma; airway innervation; C-fibers; parasympathetic neurons; neurogenic inflammation; vagal ganglia; nucleus of the solitary tract
资金
- German Center for Lung Research (DZL)
- German Research Foundation (DFG) [TRR 152 / P22]
Neuroimmune interaction has long been discussed in the pathogenesis of allergic airway diseases, such as allergic asthma. Mediators released during inflammation can alter the function of both sensory and parasympathetic neurons innervating the airways. Evidence has been provided that the inflammatory response can be altered by various mediators that are released by sensory and parasympathetic neurons and vice versa. Our aim is to demonstrate recent developments in the reciprocal neuroimmune interaction and to include, if available, data from in vivo and clinical studies.
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