相关参考文献
注意：仅列出部分参考文献，下载原文获取全部文献信息。Molecular mechanisms of protein and lipid targeting to ciliary membranes
Brian T. Emmer et al.
JOURNAL OF CELL SCIENCE (2010)
The primary cilium: a signalling centre during vertebrate development
Sarah C. Goetz et al.
NATURE REVIEWS GENETICS (2010)
Mutations in zebrafish leucine-rich repeat-containing six-like affect cilia motility and result in pronephric cysts, but have variable effects on left-right patterning
Fabrizio C. Serluca et al.
DEVELOPMENT (2009)
Dampened Hedgehog signaling but normal Wnt signaling in zebrafish without cilia
Peng Huang et al.
DEVELOPMENT (2009)
Cilia localization is essential for in vivo functions of the Joubert syndrome protein Arl13b/Scorpion
Neil A. Duldulao et al.
DEVELOPMENT (2009)
Zebrafish ift57, ift88, and ift172 Intraflagellar Transport Mutants Disrupt Cilia But Do Not Affect Hedgehog Signaling
Shannon C. Lunt et al.
DEVELOPMENTAL DYNAMICS (2009)
The Exocyst Protein Sec10 Is Necessary for Primary Ciliogenesis and Cystogenesis In Vitro
Xiaofeng Zuo et al.
MOLECULAR BIOLOGY OF THE CELL (2009)
Intraflagellar transport is required for polarized recycling of the TCR/CD3 complex to the immune synapse
Francesca Finetti et al.
NATURE CELL BIOLOGY (2009)
Strategies to inhibit cyst formation in ADPKD
James P. Calvet
CLINICAL JOURNAL OF THE AMERICAN SOCIETY OF NEPHROLOGY (2008)
Zebrafish mutations affecting cilia motility share similar cystic phenotypes and suggest a mechanism of cyst formation that differs from pkd2 morphants
Jessica Sullivan-Brown et al.
DEVELOPMENTAL BIOLOGY (2008)
Cystic kidney gene seahorse regulates cilia-mediated processes and Wnt pathways
Norihito Kishimoto et al.
DEVELOPMENTAL CELL (2008)
The mammalian Nek1 kinase is involved in primary cilium formation
Ohad Shalom et al.
FEBS LETTERS (2008)
Cyst formation and activation of the extracellular regulated kinase pathway after kidney specific inactivation of Pkd1
Sekiya Shibazaki et al.
HUMAN MOLECULAR GENETICS (2008)
Deletion of IFT20 in the mouse kidney causes misorientation of the mitotic spindle and cystic kidney disease
Julie A. Jonassen et al.
JOURNAL OF CELL BIOLOGY (2008)
The intraflagellar transport protein IFT57 is required for cilia maintenance and regulates IFT-particle-kinesin-II dissociation in vertebrate photoreceptors
Bryan L. Krock et al.
JOURNAL OF CELL SCIENCE (2008)
The subcellular localization of TRPP2 modulates its function
Xiao Fu et al.
JOURNAL OF THE AMERICAN SOCIETY OF NEPHROLOGY (2008)
High-resolution in situ hybridization to whole-mount zebrafish embryos
Christine Thisse et al.
NATURE PROTOCOLS (2008)
Genetic defects of pronephric cilia in zebrafish
Chengtian Zhao et al.
MECHANISMS OF DEVELOPMENT (2007)
Zebrafish curly up encodes a Pkd2 ortholog that restricts left-side-specific expression of southpaw
Jodi Schottenfeld et al.
DEVELOPMENT (2007)
Autosomal dominant polycystic kidney disease
Vicente E. Torres et al.
LANCET (2007)
Nodal signaling: developmental roles and regulation
Michael M. Shen
DEVELOPMENT (2007)
Molecular and cellular pathophysiology of autosomal recessive polycystic kidney disease (ARPKD)
William E. Sweeney et al.
CELL AND TISSUE RESEARCH (2006)
Polycystin-2 immunolocalization and function in zebrafish
Tomoko Obara et al.
JOURNAL OF THE AMERICAN SOCIETY OF NEPHROLOGY (2006)
The intraflagellar transport protein IFT20 is associated with the Golgi complex and is required for cilia assembly
John A. Follit et al.
MOLECULAR BIOLOGY OF THE CELL (2006)
Extracellular signal-regulated kinase inhibition slows disease progression in mice with polycystic kidney disease
Sayu Omori et al.
JOURNAL OF THE AMERICAN SOCIETY OF NEPHROLOGY (2006)
Loss of primary cilia results in deregulated and unabated apical calcium entry in ARPKD collecting duct cells
BJ Siroky et al.
AMERICAN JOURNAL OF PHYSIOLOGY-RENAL PHYSIOLOGY (2006)
Loss of polycystin-1 in human cyst-lining epithelia leads to ciliary dysfunction
Surya M. Nauli et al.
JOURNAL OF THE AMERICAN SOCIETY OF NEPHROLOGY (2006)
Calcium restores a normal proliferation phenotype in human polycystic kidney disease epithelial cells
Tamio Yamaguchi et al.
JOURNAL OF THE AMERICAN SOCIETY OF NEPHROLOGY (2006)
Polaris and Polycystin-2 in dorsal forerunner cells and Kupffer's vesicle are required for specification of the zebrafish left-right axis
BW Bisgrove et al.
DEVELOPMENTAL BIOLOGY (2005)
Large scale protein identification in intracellular aquaporin-2 vesicles from renal inner medullary collecting duct
M Barile et al.
MOLECULAR & CELLULAR PROTEOMICS (2005)
Cilia-driven fluid flow in the zebrafish pronephros, brain and Kupffer's vesicle is required for normal organogenesis
AG Kramer-Zucker et al.
DEVELOPMENT (2005)
Kupffer's vesicle is a ciliated organ of asymmetry in the zebrafish embryo that initiates left-right development of the brain, heart and gut
JJ Essner et al.
DEVELOPMENT (2005)
Pericentrin forms a complex with intraflagellar transport proteins and polycystin-2 and is required for primary cilia assembly
A Jurczyk et al.
JOURNAL OF CELL BIOLOGY (2004)
A genetic screen in zebrafish identifies cilia genes as a principal cause of cystic kidney
ZX Sun et al.
DEVELOPMENT (2004)
ERK and MMPs sequentially regulate distinct stages of epithelial tubule development
LE O'Brien et al.
DEVELOPMENTAL CELL (2004)
The exocyst localizes to the primary cilium in MDCK cells
KK Rogers et al.
BIOCHEMICAL AND BIOPHYSICAL RESEARCH COMMUNICATIONS (2004)
Intraflagellar transport genes are essential for differentiation and survival of vertebrate sensory neurons
M Tsujikawa et al.
NEURON (2004)
Two populations of node monocilia initiate left-right asymmetry in the mouse
J McGrath et al.
CELL (2003)
The exocyst affects protein synthesis by acting on the translocation machinery of the endoplasmic reticulum
JH Lipschutz et al.
JOURNAL OF BIOLOGICAL CHEMISTRY (2003)
Cyclic AMP activates B-Raf and ERK in cyst epithelial cells from autosomal-dominant polycystic kidneys
T Yamaguchi et al.
KIDNEY INTERNATIONAL (2003)
Bending the primary cilium opens Ca2+-sensitive intermediate-conductance K+ channels in MDCK cells
HA Praetorius et al.
JOURNAL OF MEMBRANE BIOLOGY (2003)
Polycystins 1 and 2 mediate mechanosensation in the primary cilium of kidney cells
SM Nauli et al.
NATURE GENETICS (2003)
The polycystic kidney disease proteins, polycystin-1, polycystin-2, polaris, and cystin, are co-localized in renal cilia
BK Yoder et al.
JOURNAL OF THE AMERICAN SOCIETY OF NEPHROLOGY (2002)
Polycystin-2 localizes to kidney cilia and the ciliary level is elevated in orpk mice with polycystic kidney disease
GJ Pazour et al.
CURRENT BIOLOGY (2002)
The ion channel polycystin-2 is required for left-right axis determination in mice
P Pennekamp et al.
CURRENT BIOLOGY (2002)
The exocyst is a Ral effector complex
S Moskalenko et al.
NATURE CELL BIOLOGY (2002)
Polaris, a protein disrupted in orpk mutant mice, is required for assembly of renal cilium
BK Yoder et al.
AMERICAN JOURNAL OF PHYSIOLOGY-RENAL PHYSIOLOGY (2002)
Bending the MDCK cell primary cilium increases intracellular calcium
HA Praetorius et al.
JOURNAL OF MEMBRANE BIOLOGY (2001)
Polycystin-2 is a novel cation channel implicated in defective intracellular Ca2+ homeostasis in polycystic kidney disease
PM Vassilev et al.
BIOCHEMICAL AND BIOPHYSICAL RESEARCH COMMUNICATIONS (2001)
Polaris, a protein involved in left-right axis patterning, localizes to basal bodies and cilia
PD Taulman et al.
MOLECULAR BIOLOGY OF THE CELL (2001)
Co-assembly of polycystin-1 and-2 produces unique cation-permeable currents
K Hanaoka et al.
NATURE (2000)
Exocyst is involved in cystogenesis and tubulogenesis and acts by modulating synthesis and delivery of basolateral plasma membrane and secretory proteins
JH Lipschutz et al.
MOLECULAR BIOLOGY OF THE CELL (2000)
Polycystin-1, the gene product of PKD1, induces resistance to apoptosis and spontaneous tubulogenesis in MDCK cells
A Boletta et al.
MOLECULAR CELL (2000)
Chlamydomonas IFT88 and its mouse homologue, polycystic kidney disease gene Tg737, are required for assembly of cilia and flagella
GJ Pazour et al.
JOURNAL OF CELL BIOLOGY (2000)
cAMP stimulates the in vitro proliferation of renal cyst epithelial cells by activating the extracellular signal-regulated kinase pathway
T Yamaguchi et al.
KIDNEY INTERNATIONAL (2000)
分享论文
