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Alisa J. Piekny et al.
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Polo-like kinase 1 triggers the initiation of cytokinesis in human cells by promoting recruitment of the RhoGEF Ect2 to the central spindle
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Choice of Plk1 docking partners during mitosis and cytokinesis is controlled by the activation state of Cdk1
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Chemical genetics reveals the requirement for Polo-like kinase 1 activity in positioning RhoA and triggering cytokinesis in human cells
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BI 2536, a potent and selective inhibitor of polo-like kinase 1, inhibits tumor growth in vivo
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Phosphorylation of the cytokinesis regulator ECT2 at G2/M phase stimulates association of the mitotic kinase Plk1 and accumulation of GTP-bound RhoA
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KIF14 and citron kinase act together to promote efficient cytokinesis
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Centralspindlin regulates ECT2 and RhoA accumulation at the equatorial cortex during cytokinesis
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Inhibition of cyclin-dependent kinase 1 induces cytokinesis without chromosome segregation in an ECT2 and MgcRacGAP-dependent manner
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An ECT2-centralspindlin complex regulates the localization and function of RhoA
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Ablation of PRC1 by small interfering RNA demonstrates that cytokinetic abscission requires a central spindle bundle in mammalian cells, whereas completion of furrowing does not
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The tandem BRCT domains of Ect2 are required for both negative and positive regulation of Ect2 in cytokinesis
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Structure and function of Polo-like kinases
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Human mitotic spindle-associated protein PRC1 inhibits MgcRacGAP activity toward Cdc42 during the metaphase
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Phosphorylation of mitotic kinesin-like protein 2 by polo-like kinase 1 is required for cytokinesis
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PRC1 is a microtubule binding and bundling protein essential to maintain the mitotic spindle midzone
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Central spindle assembly and cytokinesis require a kinesin-like protein/RhoGAP complex with microtubule bundling activity
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