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J. M. Hoenes et al.
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A variant allele of Growth Factor Independence 1 (GFI1) is associated with acute myeloid leukemia
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The Transcription Factor PU.1 Controls Dendritic Cell Development and Flt3 Cytokine Receptor Expression in a Dose-Dependent Manner
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HOXA9 is required for survival in human MLL-rearranged acute leukemias
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Gfi1 integrates progenitor versus granulocytic transcriptional programming
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HAEMATOLOGICA-THE HEMATOLOGY JOURNAL (2009)
Growth factor independent 1b (Gfi1b) and a new splice variant of Gfi1b are highly expressed in patients with acute and chronic leukemia
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INTERNATIONAL JOURNAL OF HEMATOLOGY (2009)
Molecular prognostic markers for adult acute myeloid leukemia with normal cytogenetics
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Gfi1b:: green fluorescent protein knock-in mice reveal a dynamic expression pattern of Gfi1b during hematopoiesis that is largely complementary to Gfi1
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Meis1 programs transcription of FLT3 and cancer stem cell character, using a mechanism that requires interaction with Pbx and a novel function of the Meis1 C-terminus
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Gfi-1 restricts proliferation and preserves functional integrity of haematopoietic stem cells
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Transformation of myeloid progenitors by MLL oncoproteins is dependent on Hoxa7 and Hoxa9
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Mutations in proto-oncogene GFI1 cause human neutropenia and target ELA2
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FLT3 internal tandem duplication mutations associated with human acute myeloid leukemias induce myeloproliferative disease in a murine bone marrow transplant model
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The presence of a FLT3 internal tandem duplication in patients with acute myeloid leukemia (AML) adds important prognostic information to cytogenetic risk group and response to the first cycle of chemotherapy: analysis of 854 patients from the United Kingdom Medical Research Council AML 10 and 12 trials
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