期刊
TRENDS IN CELL BIOLOGY
卷 18, 期 1, 页码 1-4出版社
ELSEVIER SCIENCE LONDON
DOI: 10.1016/j.tcb.2007.11.003
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Disruption of protein homeostasis in mitochondria elicits a cellular response, which upregulates mitochondrial chaperones and other factors that serve to remodel the mitochondrial-folding environment. In a recent study, Haynes and colleagues uncovered a novel signal transduction pathway underlying this process. The upstream mitochondrial component of this pathway is an orthologue of Escherichia coli CIpP, which functions in the bacterial heat-shock response. These findings suggest that molecular aspects of stress sensing might be conserved between bacteria and mitochondria.
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