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Effects of prenatal and postnatal exposure to a low dose of bisphenol A on behavior and memory in rats
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Perinatal exposure to bisphenol-A impairs learning-memory by concomitant down-regulation of N-methyl-D-aspartate receptors of hippocampus in male offspring mice
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Neonatal exposure to endocrine active compounds or an ER agonist increases adult anxiety and aggression in gonadally intact male rats
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Bisphenol A prevents the synaptogenic response to estradiol in hippocampus and prefrontal cortex of ovariectomized nonhuman primates
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No effect of route of exposure (oral; subcutaneous injection) on plasma bisphenol A throughout 24 h after administration in neonatal female mice
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Evidence of altered brain sexual differentiation in mice exposed perinatally to low, environmentally relevant levels of bisphenol A
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Prenatal exposure to bisphenol A impairs sexual differentiation of exploratory behavior and increases depression-like behavior in rats
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An extensive new literature concerning low-dose effects of bisphenol A shows the need for a new risk assessment
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The environmental estrogen bisphenol a inhibits estradiol-induced hippocampal synaptogenesis
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The 17 alpha and 17 beta isomers of estradiol both induce rapid spine synapse formation in the CA1 hippocampal subfield of ovariectomized female rats
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