相关参考文献
注意：仅列出部分参考文献，下载原文获取全部文献信息。Anti-apoptotic Mcl-1 is essential for the development and sustained growth of acute myeloid leukemia
Stefan P. Glaser et al.
GENES & DEVELOPMENT (2012)
Substantial Susceptibility of Chronic Lymphocytic Leukemia to BCL2 Inhibition: Results of a Phase I Study of Navitoclax in Patients With Relapsed or Refractory Disease
Andrew W. Roberts et al.
JOURNAL OF CLINICAL ONCOLOGY (2012)
Anti-apoptotic MCL-1 localizes to the mitochondrial matrix and couples mitochondrial fusion to respiration
Rhonda M. Perciavalle et al.
NATURE CELL BIOLOGY (2012)
Deciphering the rules of programmed cell death to improve therapy of cancer and other diseases
Andreas Strasser et al.
EMBO JOURNAL (2011)
SCFFBW7 regulates cellular apoptosis by targeting MCL1 for ubiquitylation and destruction
Hiroyuki Inuzuka et al.
NATURE (2011)
Sensitivity to antitubulin chemotherapeutics is regulated by MCL1 and FBW7
Ingrid E. Wertz et al.
NATURE (2011)
Elevated Mcl-1 perturbs lymphopoiesis, promotes transformation of hematopoietic stem/progenitor cells, and enhances drug resistance
Kirsteen J. Campbell et al.
BLOOD (2010)
The fast-mobility isoform of mouse Mcl-1 is a mitochondrial matrix-localized protein with attenuated anti-apoptotic activity
Chi-Ruei Huang et al.
FEBS LETTERS (2010)
Mcl1 haploinsufficiency protects mice from Myc-induced acute myeloid leukemia
Zhifu Xiang et al.
JOURNAL OF CLINICAL INVESTIGATION (2010)
Ubiquitin-Independent Degradation of Antiapoptotic MCL-1
Daniel P. Stewart et al.
MOLECULAR AND CELLULAR BIOLOGY (2010)
Deubiquitinase USP9X stabilizes MCL1 and promotes tumour cell survival
Martin Schwickart et al.
NATURE (2010)
The landscape of somatic copy-number alteration across human cancers
Rameen Beroukhim et al.
NATURE (2010)
Mitochondria and cell death: outer membrane permeabilization and beyond
Stephen W. G. Tait et al.
NATURE REVIEWS MOLECULAR CELL BIOLOGY (2010)
Mcl-1 Is Essential for Germinal Center Formation and B Cell Memory
Ingela Vikstrom et al.
SCIENCE (2010)
Male germ cell development: turning on the apoptotic pathways
Rakshamani Tripathi et al.
JOURNAL OF REPRODUCTIVE IMMUNOLOGY (2009)
Loss of BH3-only protein bim inhibits apoptosis of hemopoietic cells in the fetal liver and male germ cells but not neuronal cells in Bcl-x-deficient mice
Rizwan S. Akhtar et al.
JOURNAL OF HISTOCHEMISTRY & CYTOCHEMISTRY (2008)
BCL-2 family antagonists for cancer therapy
Guillaume Lessene et al.
NATURE REVIEWS DRUG DISCOVERY (2008)
N-terminal truncation of antiapoptotic MCL1, but not G2/M-induced phosphorylation, is associated with stabilization and abundant expression in tumor cells
Alfredo De Biasio et al.
JOURNAL OF BIOLOGICAL CHEMISTRY (2007)
Degradation of Mcl-1 by β-TrCP mediates glycogen synthase kinase 3-induced tumor suppression and chemosensitization
Qingqing Ding et al.
MOLECULAR AND CELLULAR BIOLOGY (2007)
Programmed anuclear cell death delimits platelet life span
Kylie D. Mason et al.
CELL (2007)
The BH3 mimetic ABT-737 targets selective Bcl-2 proteins and efficiently induces apoptosis via Bak/Bax if Mcl-1 is neutralized
Mark F. van Delft et al.
CANCER CELL (2006)
Concomitant loss of proapoptotic BH3-only bcl-2 antagonists Bik and Bim arrests spermatogenesis
L Coultas et al.
EMBO JOURNAL (2005)
Mule/ARF-BP1, a BH3-only E3 ubiquitin ligase, catalyzes the polyubiquitination of Mcl-1 and regulates apoptosis
Q Zhong et al.
CELL (2005)
Proapoptotic Bak is sequestered by Mcl-1 and Bcl-xL, but not Bcl-2, until displaced by BH3-only proteins
SN Willis et al.
GENES & DEVELOPMENT (2005)
Obligate role of anti-apoptotic MCL-1 in the survival of hematopoietic stem cells
JT Opferman et al.
SCIENCE (2005)
DNA damage response and MCL-1 destruction initiate apoptosis in adenovirus-infected cells
A Cuconati et al.
GENES & DEVELOPMENT (2003)
Elimination of Mcl-1 is required for the initiation of apoptosis following ultraviolet irradiation
D Nijhawan et al.
GENES & DEVELOPMENT (2003)
Bcl-x and bax regulate mouse primordial germ cell survival and apoptosis during embryogenesis
EB Rucker et al.
MOLECULAR ENDOCRINOLOGY (2000)
分享论文
