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Paolo Pinton et al.
CURRENT MOLECULAR MEDICINE (2008)
被撤回的出版物: Fhit interaction with ferredoxin reductase triggers generation of reactive oxygen species and apoptosis of cancer cells (Retracted article. See vol. 292, pg. 14279, 2017)
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Calcium and apoptosis: ER-mitochondria Ca2+ transfer in the control of apoptosis
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Phosphorylation of inositol 1,4,5-trisphosphate receptors by protein kinase B/Akt inhibits Ca2+ release and apoptosis
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The mitochondrial permeability transition from in vitro artifact to disease target
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Inhibitory interaction of the 14-3-3ε protein with isoform 4 of the plasma membrane Ca2+-ATPase pump
A Rimessi et al.
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The endoplasmic reticulum gateway to apoptosis by Bcl-XL modulation of the InsP3R
C White et al.
NATURE CELL BIOLOGY (2005)
Components of DNA damage checkpoint pathway regulate UV exposure-dependent alterations of gene expression of FHIT and WWOX at chromosome fragile sites
H Ishii et al.
MOLECULAR CANCER RESEARCH (2005)
Cleavage of the plasma membrane Na+/Ca2+ exchanger in excitotoxicity
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CELL (2005)
Loss of FHIT protein expression is related to high proliferation, low apoptosis and worse prognosis in non-small-cell lung cancer
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Drp-1-dependent division of the mitochondrial network blocks intraorganellar Ca2+ waves and protects against Ca2+-mediated apoptosis
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MOLECULAR CELL (2004)
Stable interactions between mitochondria and endoplasmic reticulum allow rapid accumulation of calcium in a subpopulation of mitochondria
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Caspase-dependent alterations of Ca2+ signaling in the induction of apoptosis by hepatitis B virus X protein
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NATURE REVIEWS MOLECULAR CELL BIOLOGY (2003)
Cancer and the FRA3B/FHIT fragile locus: it's a HIT
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BAX and BAK regulation of endoplasmic reticulum Ca2+:: A control point for apoptosis
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SCIENCE (2003)
Recombinant expression of the voltage-dependent anion channel enhances the transfer of Ca2+ microdomains to mitochondria
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Hint, Fhit, and GalT: Function, structure, evolution, and mechanism of three branches of the histidine triad superfamily of nucleotide hydrolases and transferases
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BIOCHEMISTRY (2002)
FHIT as tumor suppressor:: Mechanisms and therapeutic opportunities
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CANCER BIOLOGY & THERAPY (2002)
Restoration of fragile histidine triad (FHIT) expression induces apoptosis and suppresses tumorigenicity in lung and cervical cancer cell lines
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FRA3B and other common fragile sites:: The weakest links
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PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA (2001)
Propagation of the apoptotic signal by mitochondrial waves
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EMBO JOURNAL (2001)
The Ca2+ concentration of the endoplasmic reticulum is a key determinant of ceramide-induced apoptosis:: significance for the molecular mechanism of Bcl-2 action
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EMBO JOURNAL (2001)
FHIT gene therapy prevents tumor development in Fhit-deficient mice
KR Dumon et al.
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Respiring mitochondria determine the pattern of activation and inactivation of the store-operated Ca2+ current ICRAC
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EMBO JOURNAL (2000)
Mitochondria and calcium: from cell signalling to cell death
MR Duchen
JOURNAL OF PHYSIOLOGY-LONDON (2000)
Reduced loading of intracellular Ca2+ stores and downregulation of capacitative Ca2+ influx in Bcl-2-overexpressing cells
P Pinton et al.
JOURNAL OF CELL BIOLOGY (2000)