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Hypoxia-induced endothelial proliferation requires both mTORC1 and mTORC2
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Cdc2-cyclin E complexes regulate the G1/S phase transition
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Cdk2 is dispensable for cell cycle inhibition and tumor suppression mediated by p27Kip1 and p21Cip1
A Martín et al.
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Mouse development and cell proliferation in the absence of D-cyclins
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M Malumbres et al.
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An Rb-Skp2-p27 pathway mediates acute cell cycle inhibition by Rb and is retained in a partial-penetrance Rb mutant
P Ji et al.
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Cdk2 knockout mice are viable
C Berthet et al.
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Oxygen sensitivity severely limits the replicative lifespan of murine fibroblasts
S Parrinello et al.
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Acute mutation of retinoblastoma gene function is sufficient for cell cycle re-entry
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Extra-embryonic function of Rb is essential for embryonic development and viability
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Targeted disruption of the three Rb-related genes leads to loss of G1 control and immortalization
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Ablation of the Retinoblastoma gene family deregulates G1 control causing immortalization and increased cell turnover under growth-restricting conditions
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