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Anthony Davies et al.
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Kinetics of internalization and degradation of N-type voltage-gated calcium channels:: Role of the α2/δ subunit
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Identification of the α2-δ-1 subunit of voltage-dependent calcium channels as a molecular target for pain mediating the analgesic actions of pregabalin
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Pregabalin reduces the release of synaptic vesicles from cultured hippocampal neurons
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The metal-ion-dependent adhesion site in the Von Willebrand factor-A domain of α2δ subunits is key to trafficking voltage-gated Ca2+ channels
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PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA (2005)
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β subunits of voltage-gated calcium channels
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JOURNAL OF BIOENERGETICS AND BIOMEMBRANES (2003)
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Pregabalin and gabapentin reduce release of substance P and CGRP from rat spinal tissues only after inflammation or activation of protein kinase C
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Gabapentin inhibits presynaptic Ca2+ influx and synaptic transmission in rat hippocampus and neocortex
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Gabapentin-mediated inhibition of voltage-activated Ca2+ channel currents in cultured sensory neurones is dependent on culture conditions and channel subunit expression
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