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Mitochondrial pathology and muscle and dopaminergic neuron degeneration caused inactivation of Drosophila Pink1 is rescued by by Parkin
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Drosophila pink1 is required for mitochondrial function and interacts genetically with parkin
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Drosophila parkin mutants have decreased mass and cell size and increased sensitivity to oxygen radical stress
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The mitochondrial protein hFis1 regulates mitochondrial fission in mammalian cells through an interaction with the dynamin-like protein DLP1
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Parkin suppresses dopaminergic neuron-selective neurotoxicity induced by Pael-R in Drosophila
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The WD repeat protein, Mdv1p, functions as a molecular adaptor by interacting with Dnm1p and Fis1p during mitochondrial fission
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