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Disruption of planar cell polarity signaling results in congenital heart defects and cardiomyopathy attributable to early cardiomyocyte disorganization
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HUMAN MOLECULAR GENETICS (2006)
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Erosive vitreoretinopathy and Wagner disease are caused by intronic mutations in CSPG2/Versican that result in an imbalance of splice variants
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Proteolytic cleavage of versican during cardiac cushion morphogenesis
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JOURNAL OF ALLERGY AND CLINICAL IMMUNOLOGY (2006)
Neural crest cells retain multipotential characteristics in the developing valves and label the cardiac conduction system
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Versican mediates mesenchymal-epithelial transition
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Annexin A6 stimulates the membrane recruitment of p120GAP to modulate Ras and Raf-1 activity
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Vangl2 acts via RhoA signaling to regulate polarized cell movements during development of the proximal outflow tract
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Do cypher gene mutations cause left ventricular noncompaction with subdinical myopathy
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CIRCULATION RESEARCH (2004)
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Mutations in Cypher/ZASP in patients with dilated cardiomyopathy and left ventricular non-compaction
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JOURNAL OF THE AMERICAN COLLEGE OF CARDIOLOGY (2003)
Versican: a versatile extracellular matrix proteoglycan in cell biology
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Ablation of Cypher, a PDZ-LIM domain Z-line protein, causes a severe form of congenital myopathy
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Versican V1 proteolysis in human aorta in vivo occurs at the Glu441-Ala442 bond, a site that is cleaved by recombinant ADAMTS-1 and ADAMTS-4
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