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Liang Wei et al.
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Changing Mad2 Levels Affects Chromosome Segregation and Spindle Assembly Checkpoint Control in Female Mouse Meiosis I
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The spindle-assembly checkpoint in space and time
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Cytoplasmic dynein participates in meiotic checkpoint inactivation in mouse oocytes by transporting cytoplasmic mitotic arrest-deficient (Mad) proteins from kinetochores to spindle poles
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Mad2 is required for inhibiting securin and cyclin B degradation following spindle depolymerisation in meiosis I mouse oocytes
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Increased chromosome instability but not cancer predisposition in haploinsufficient Bub3 mice
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PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA (2005)
Mad2 prevents aneuploidy and premature proteolysis of cyclin B and securin during meiosis I in mouse oocytes
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PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA (2004)
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The spindle assembly checkpoint is not essential for CSF arrest of mouse oocytes
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JOURNAL OF CELL BIOLOGY (2004)
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JOURNAL OF CELL BIOLOGY (2003)
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JOURNAL OF CELL BIOLOGY (2000)
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