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注意:仅列出部分参考文献,下载原文获取全部文献信息。Stem Cell Factor SALL4 Represses the Transcriptions of PTEN and SALL1 through an Epigenetic Repressor Complex
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Qing Zhou et al.
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AMERICAN JOURNAL OF MEDICAL GENETICS PART A (2007)
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Murine inner cell mass-derived lineages depend on SaII4 function
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SALL4, a novel oncogene, is constitutively expressed in human acute myeloid leukemia (AML) and induces AML in transgenic mice
Yupo Ma et al.
BLOOD (2006)
Sall4 modulates embryonic stem cell pluripotency and early embryonic development by the transcriptional regulation of Pou5f1
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Sall4 interacts with nanog and co-occupies nanog genomic sites in embryonic stem cells
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The murine homolog of SALL4, a causative gene in Okihiro syndrome, is essential for embryonic stem cell proliferation, and cooperates with Sall1 in anorectal, heart, brain and kidney development
Masayo Sakaki-Yumoto et al.
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Transcriptional activation of the SALL1 by the human SIX1 homeodomain during kidney development
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The Oct4 and Nanog transcription network regulates pluripotency in mouse embryonic stem cells
YH Loh et al.
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SALL4 mutations Okihiro syndrome (Duane-radial ray syndrome), acro-renal-ocular syndrome, and related disorders
J Kohlhase et al.
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Identification, cloning and expression analysis of the pluripotency promoting Nanog genes in mouse and human
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Loss of the Sall3 gene leads to palate deficiency, abnormalities in cranial nerves, and perinatal lethality
M Parrish et al.
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Mutations at the SALL4 locus on chromosome 20 result in a range of clinically overlapping phenotypes, including Okihiro syndrome, Holt-Oram syndrome, acro-renal-ocular syndrome, and patients previously reported to represent thalidomide embryopathy
J Kohlhase et al.
JOURNAL OF MEDICAL GENETICS (2003)
Duane radial ray syndrome (Okihiro syndrome) maps to 20q13 and results from mutations in SALL4, a new member of the SAL family
R Al-Baradie et al.
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