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注意：仅列出部分参考文献，下载原文获取全部文献信息。STAT1 and STAT3 do not participate in FGF-mediated growth arrest in chondrocytes
Pavel Krejci et al.
JOURNAL OF CELL SCIENCE (2008)
Sustained phosphorylation of mutated FGFR3 is a crucial feature of genetic dwarfism and induces apoptosis in the ATDC5 chondrogenic cell line via PLCγ-activated STATI
Daisuke Harada et al.
BONE (2007)
Bisindolylmaleimide I suppresses fibroblast growth factor-mediated activation of Erk MAP kinase in chondrocytes by preventing Shp2 association with the Frs2 and Gab1 adaptor proteins
Pavel Krejci et al.
JOURNAL OF BIOLOGICAL CHEMISTRY (2007)
Interaction of fibroblast growth factor and C-natriuretic peptide signaling in regulation of chondrocyte proliferation and extracellular matrix homeostasis
P Krejci et al.
JOURNAL OF CELL SCIENCE (2005)
Sustained ERK1/2 but not STAT1 or 3 activation is required for thanatophoric dysplasia phenotypes in PC12 cells
N Nowroozi et al.
HUMAN MOLECULAR GENETICS (2005)
FGF signaling in the developing endochondral skeleton
DM Ornitz
CYTOKINE & GROWTH FACTOR REVIEWS (2005)
Cell responses to FGFR3 signalling:: growth, differentiation and apoptosis
CGM L'Hôte et al.
EXPERIMENTAL CELL RESEARCH (2005)
Constitutive activation of MEK1 in chondrocytes causes Stat1-independent achondroplasia-like dwarfism and rescues the Fgfr3-deficient mouse phenotype
S Murakami et al.
GENES & DEVELOPMENT (2004)
Activation of the ERK1/2 and p38 mitogen-activated protein kinase pathways mediates fibroblast growth factor-induced growth arrest of chondrocytes
A Raucci et al.
JOURNAL OF BIOLOGICAL CHEMISTRY (2004)
FGF2 inhibits proliferation and alters the cartilage-like phenotype of RCS cells
P Krejci et al.
EXPERIMENTAL CELL RESEARCH (2004)
Overexpression of CNP in chondrocytes rescues achondroplasia through a MAPK-dependent pathway
A Yasoda et al.
NATURE MEDICINE (2004)
Overexpression of FGFR3, Stat1, Stat5 and p21(Cip1) correlates with phenotypic severity and defective chondrocyte differentiation in FGFR3-related chondrodysplasias
L Legeai-Mallet et al.
BONE (2004)
The kinase activity of fibroblast growth factor receptor 3 with activation loop mutations affects receptor trafficking and signaling
PMJ Lievens et al.
JOURNAL OF BIOLOGICAL CHEMISTRY (2004)
A network of transcriptional and signaling events is activated by FGF to induce chondrocyte growth arrest and differentiation
L Dailey et al.
JOURNAL OF CELL BIOLOGY (2003)
The thanatophoric dysplasia type II mutation hampers complete maturation of fibroblast growth factor receptor 3 (FGFR3), which activates signal transducer and activator of transcription 1 (STAT1) from the endoplasmic reticulum
PMJ Lievens et al.
JOURNAL OF BIOLOGICAL CHEMISTRY (2003)
Fibroblast growth factor inhibits chondrocytic growth through induction of p21 and subsequent inactivation of cyclin E-Cdk2
T Aikawa et al.
JOURNAL OF BIOLOGICAL CHEMISTRY (2001)
Deregulated FGFR3 mutants in multiple myeloma cell lines with t(4;14): comparative analysis of Y373C, K650E and the novel G384D mutations
D Ronchetti et al.
ONCOGENE (2001)
Identification of tyrosine residues in constitutively activated fibroblast growth factor receptor 3 involved in mitogenesis, Stat activation, and phosphatidylinositol 3-kinase activation
KC Hart et al.
MOLECULAR BIOLOGY OF THE CELL (2001)
Transformation and Stat activation by derivatives of FGFR1, FGFR3, and FGFR4
KC Hart et al.
ONCOGENE (2000)
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