期刊
PHYSIOLOGY
卷 24, 期 6, 页码 357-366出版社
AMER PHYSIOLOGICAL SOC
DOI: 10.1152/physiol.00029.2009
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- RIKEN
- National Institute on Drug Abuse [R01 DA-17310]
- Ministry of Education, Science, and Culture of Japan
- Generalitat de Catalunya, Spain
Ca2+/calmodulin-dependent protein kinase II (CaMKII) and actin are two crucial molecules involved in long-term potentiation (LTP). In addition to its signaling function, CaMKII plays a structural role via direct interaction with actin filaments, thus coupling functional and structural plasticity in dendritic spines. The status of F-actin, regulated by CaMKII, determines the postsynaptic protein binding capacity and thus may act as a synaptic tag that consolidates LTP.
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