相关参考文献
注意:仅列出部分参考文献,下载原文获取全部文献信息。Translocation of iron from lysosomes to mitochondria during ischemia predisposes to injury after reperfusion in rat hepatocytes
Xun Zhang et al.
FREE RADICAL BIOLOGY AND MEDICINE (2013)
Cellular-Specific Role of Toll-Like Receptor 4 in Hepatic Ischemia-Reperfusion Injury in Mice
Gary W. Nace et al.
HEPATOLOGY (2013)
The role of intracellular high-mobility group box 1 in the early activation of Kupffer cells and the development of Con A-induced acute liver failure
Qiao Yang et al.
IMMUNOBIOLOGY (2013)
Molecular mechanisms of liver ischemia reperfusion injury: Insights from transgenic knockout models
Gourab Datta et al.
WORLD JOURNAL OF GASTROENTEROLOGY (2013)
The sterile immune response during hepatic ischemia/reperfusion
Rowan F. van Golen et al.
CYTOKINE & GROWTH FACTOR REVIEWS (2012)
Current strategies to minimize hepatic ischemia-reperfusion injury by targeting reactive oxygen species
Hartmut Jaeschke et al.
TRANSPLANTATION REVIEWS (2012)
Tumor Necrosis Factor-α in Liver Ischemia/Reperfusion Injury
Brandon C. Perry et al.
JOURNAL OF INVESTIGATIVE SURGERY (2011)
The Role of HMGB1 and HSP72 in Ischemia and Reperfusion Injury
Wilson J. Chang et al.
JOURNAL OF SURGICAL RESEARCH (2011)
High Mobility Group Box 1 Release from Hepatocytes during Ischemia and Reperfusion Injury Is Mediated by Decreased Histone Deacetylase Activity
John Evankovich et al.
JOURNAL OF BIOLOGICAL CHEMISTRY (2010)
Intracellular Calcium Signaling Pathways during Liver Ischemia and Reperfusion
Wilson J. Chang et al.
JOURNAL OF INVESTIGATIVE SURGERY (2010)
Ischemic Preconditioning (IP) of the Liver as a Safe and Protective Technique against Ischemia/Reperfusion Injury (IRI)
A. Franchello et al.
AMERICAN JOURNAL OF TRANSPLANTATION (2009)
How mitochondria produce reactive oxygen species
Michael P. Murphy
BIOCHEMICAL JOURNAL (2009)
CD4 T Cells Promote Tissue Inflammation via CD40 Signaling Without De Novo Activation in a Murine Model of Liver Ischemia/Reperfusion Injury
Xiuda Shen et al.
HEPATOLOGY (2009)
Searching for NF-kappa B-Based Treatments of Ischemia Reperfusion Injury
Christopher A. Latanich et al.
JOURNAL OF INVESTIGATIVE SURGERY (2009)
A mitochondria-targeted S-nitrosothiol modulates respiration, nitrosates thiols, and protects against ischemia-reperfusion injury
Tracy A. Prime et al.
PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA (2009)
Reciprocal activation between CD4+ T cells and Kupffer cells during hepatic ischemia-reperfusion
Marc Hanschen et al.
TRANSPLANTATION (2008)
HMGB1 release induced by liver ischemia involves Toll-like receptor 4-dependent reactive oxygen species production and calcium-mediated signaling
Allan Tsung et al.
JOURNAL OF EXPERIMENTAL MEDICINE (2007)
ROS scavenging before 27°C ischemia protects hearts and reduces mitochondrial ROS, Ca2+ overload, and changes in redox state
Amadou K. S. Camara et al.
AMERICAN JOURNAL OF PHYSIOLOGY-CELL PHYSIOLOGY (2007)
How do enzymes activate oxygen without inactivating themselves?
Judith P. Klinman
ACCOUNTS OF CHEMICAL RESEARCH (2007)
Oxygen sensing by mitochondria at complex III: the paradox of increased reactive oxygen species during hypoxia
Robert D. Guzy et al.
EXPERIMENTAL PHYSIOLOGY (2006)
The mechanism of superoxide production by NADH:ubiquinone oxidoreductase (complex I) from bovine heart mitochondria
Lothar Kussmaul et al.
PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA (2006)
NO restores HIF-1α hydroxylation during hypoxia:: Role of reactive oxygen species
M Callapina et al.
FREE RADICAL BIOLOGY AND MEDICINE (2005)
A targeted antioxidant reveals the importance of mitochondrial reactive oxygen species in the hypoxic signaling of HIF-1α
A Sanjuán-Pla et al.
FEBS LETTERS (2005)
The nuclear factor HMGB1 mediates hepatic injury after murine liver ischemia-reperfusion
A Tsung et al.
JOURNAL OF EXPERIMENTAL MEDICINE (2005)
O2-regulated gene expression:: transcriptional control of cardiorespiratory physiology by HIF-1
GL Semenza
JOURNAL OF APPLIED PHYSIOLOGY (2004)
Reduced reactive O2 species formation and preserved mitochondrial NADH and [Ca2+] levels during short-term 17 °C ischemia in intact hearts
ML Riess et al.
CARDIOVASCULAR RESEARCH (2004)
Superoxide production by NADH: ubiquinone oxidoreductase (complex I) depends on the pH gradient across the mitochondrial inner membrane
AJ Lambert et al.
BIOCHEMICAL JOURNAL (2004)
Susceptibility of murine periportal hepatocytes to hypoxia-reoxygenation: Role for NO and Kupffer cell-derived oxidants
H Taniai et al.
HEPATOLOGY (2004)
Differential roles of hypoxia-inducible factor 1α (HIF-1α) and HIF-2α in hypoxic gene regulation
CJ Hu et al.
MOLECULAR AND CELLULAR BIOLOGY (2003)
Mitochondrial formation of reactive oxygen species
JF Turrens
JOURNAL OF PHYSIOLOGY-LONDON (2003)
Apoptosis versus oncotic necrosis in hepatic ischemia/reperfusion injury
H Jaeschke et al.
GASTROENTEROLOGY (2003)
Role of the mitochondrial permeability transition in apoptotic and necrotic death after ischemia/reperfusion injury to hepatocytes
JS Kim et al.
CURRENT MOLECULAR MEDICINE (2003)
Transferrin receptor gene expression and transferrin-bound iron uptake are increased during postischemic rat liver reperfusion
L Tacchini et al.
HEPATOLOGY (2002)
Role of oxidants in NF-κB activation and TNF-α gene transcription induced by hypoxia and endotoxin
NS Chandel et al.
JOURNAL OF IMMUNOLOGY (2000)
Smac, a mitochondrial protein that promotes cytochrome c-dependent caspase activation by eliminating IAP inhibition
CY Du et al.
CELL (2000)
Identification of DIABLO, a mammalian protein that promotes apoptosis by binding to and antagonizing IAP proteins
AM Verhagen et al.
CELL (2000)