期刊
PFLUGERS ARCHIV-EUROPEAN JOURNAL OF PHYSIOLOGY
卷 460, 期 2, 页码 437-450出版社
SPRINGER HEIDELBERG
DOI: 10.1007/s00424-010-0788-2
关键词
TRP channels; Channelopathies; Drug targets; Hereditary diseases; SNP; Cation channel; Calcium signalling; Calcium; Magnesium
类别
资金
- Belgian Ministry for Science Policy [IUAP P6/28]
- Research Foundation-Flanders [G.0172.03, G.0565.07]
- Research Council of the KU Leuven [GOA 2004/07, EF/95/010]
In the past years, several hereditary diseases caused by defects in transient receptor potential channels (TRP) genes have been described. This review summarizes our current knowledge about TRP channelopathies and their possible pathomechanisms. Based on available genetic indications, we will also describe several putative pathological conditions in which (mal)function of TRP channels could be anticipated.
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