期刊
TRENDS IN MOLECULAR MEDICINE
卷 21, 期 12, 页码 731-733出版社
ELSEVIER SCI LTD
DOI: 10.1016/j.molmed.2015.10.006
关键词
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Improving the limited ability of the heart to regenerate after infarction is crucial. Researchers now demonstrate that delivery of follistatin-like 1 (FSTL1) into injured hearts via collagen patches stimulates cardiomyocyte proliferation and cardiac functional recovery. These findings highlight the epicardium as a source of novel regenerative factors and biomimetic nanomaterials in cardiac translational medicine.
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