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A splice variant of the Wilms' tumour suppressor Wt1 is required for normal development of the olfactory system
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The Wt1+/R394W mouse displays glomerulosclerosis and early-onset renal failure characteristic of human Denys-Drash syndrome
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The Wilms' tumour suppressor protein, WT1, undergoes CRM1-independent nucleocytoplasmic shuttling
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Mice lacking the 68-amino-acid, mammal-specific N-terminal extension of WT1 develop normally and are fertile
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A mutant form of the Wilms' tumor suppressor gene WT1 observed in Denys-Drash syndrome interferes with glomerular capillary development
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