相关参考文献
注意:仅列出部分参考文献,下载原文获取全部文献信息。Hypoxia-Inducible Factors in Physiology and Medicine
Gregg L. Semenza
CELL (2012)
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HIF-2α, but not HIF-1α, promotes iron absorption in mice
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Poly(ADP-ribose) polymerase 1 promotes tumor cell survival by coactivating hypoxia-inducible factor-1-dependent gene expression
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HIF-2α promotes hypoxic cell proliferation by enhancing c-Myc transcriptional activity
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Hypoxia-inducible factor-2 (HIF-2) regulates hepatic erythropoietin in vivo
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Modulation of transcription by PARP-1:: Consequences in carcinogenesis and inflammation
R. Aguilar-Quesada et al.
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Recruitment of HIF-1α and HIF-2α to common target genes is differentially regulated in neuroblastoma:: HIF-2α promotes an aggressive phenotype
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HIF-2α regulates Oct-4:: effects of hypoxia on stem cell function, embryonic development, and tumor growth
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Identification of a functional hypoxia-responsive element that regulates the expression of the egl nine homologue 3 (egln3/phd3) gene
N Pescador et al.
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Contrasting properties of hypoxia-inducible factor 1 (HIF-1) and HIF-2 in von Hippel-Lindau-associated renal cell carcinoma
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HIF-1α induces cell cycle arrest by functionally counteracting Myc
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HIF prolyl-hydroxylase 2 is the key oxygen sensor setting low steady-state levels of HIF-1α in normoxia
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Inhibition of HIF2 alpha is sufficient to suppress pVHL-defective tumor growth
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Expression of hypoxia-inducible factor-1α and -2α in hypoxic and ischemic rat kidneys
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FIH-1 is an asparaginyl hydroxylase enzyme that regulates the transcriptional activity of hypoxia-inducible factor
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The contribution of VHL substrate binding and HIF1-α to the phenotype of VHL loss in renal cell carcinoma
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Inhibition of HIF is necessary for tumor suppression by the von Hippel-Lindau protein
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A conserved family of prolyl-4-hydroxylases that modify HIF
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C-elegans EGL-9 and mammalian homologs define a family of dioxygenases that regulate HIF by prolyl hydroxylation
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CELL (2001)
HIFα targeted for VHL-mediated destruction by proline hydroxylation:: Implications for O2 sensing
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Targeting of HIF-α to the von Hippel-Lindau ubiquitylation complex by O2-regulated prolyl hydroxylation
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The transcription factor EPAS-1/hypoxia-inducible factor 2α plays an important role in vascular remodeling
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