相关参考文献
注意:仅列出部分参考文献,下载原文获取全部文献信息。A Distinct Replication Fork Protection Pathway Connects Fanconi Anemia Tumor Suppressors to RAD51-BRCA1/2
Katharina Schlacher et al.
CANCER CELL (2012)
RMI1 Promotes DNA Replication Fork Progression and Recovery from Replication Fork Stress
Jay Yang et al.
MOLECULAR AND CELLULAR BIOLOGY (2012)
Fanconi anemia proteins FANCD2 and FANCI exhibit different DNA damage responses during S-phase
Archana Sareen et al.
NUCLEIC ACIDS RESEARCH (2012)
Double-Strand Break Repair-Independent Role for BRCA2 in Blocking Stalled Replication Fork Degradation by MRE11
Katharina Schlacher et al.
CELL (2011)
Interaction between the helicases genetically linked to Fanconi anemia group J and Bloom's syndrome
Avvaru N. Suhasini et al.
EMBO JOURNAL (2011)
SLX4, a coordinator of structure-specific endonucleases, is mutated in a new Fanconi anemia subtype
Chantal Stoepker et al.
NATURE GENETICS (2011)
Disruption of mouse Slx4, a regulator of structure-specific nucleases, phenocopies Fanconi anemia
Gerry P. Crossan et al.
NATURE GENETICS (2011)
Mutations of the SLX4 gene in Fanconi anemia
Yonghwan Kim et al.
NATURE GENETICS (2011)
Involvement of SLX4 in interstrand cross-link repair is regulated by the Fanconi anemia pathway
Kimiyo N. Yamamoto et al.
PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA (2011)
Pyrimidine pool imbalance induced by BLM helicase deficiency contributes to genetic instability in Bloom syndrome
Pauline Chabosseau et al.
NATURE COMMUNICATIONS (2011)
Identification of KIAA1018/FAN1, a DNA Repair Nuclease Recruited to DNA Damage by Monoubiquitinated FANCD2
Craig MacKay et al.
CELL (2010)
Deficiency of FANCD2-Associated Nuclease KIAA1018/FAN1 Sensitizes Cells to Interstrand Crosslinking Agents
Katja Kratz et al.
CELL (2010)
A novel role for fanconi anemia (FA) pathway effector protein FANCD2 in cell cycle progression of untransformed primary human cells
Ihn Young Song et al.
CELL CYCLE (2010)
Expanded roles of the Fanconi anemia pathway in preserving genomic stability
Younghoon Kee et al.
GENES & DEVELOPMENT (2010)
Hydroxyurea-Stalled Replication Forks Become Progressively Inactivated and Require Two Different RAD51-Mediated Pathways for Restart and Repair
Eva Petermann et al.
MOLECULAR CELL (2010)
A Genetic Screen Identifies FAN1, a Fanconi Anemia-Associated Nuclease Necessary for DNA Interstrand Crosslink Repair
Agata Smogorzewska et al.
MOLECULAR CELL (2010)
Pathways of mammalian replication fork restart
Eva Petermann et al.
NATURE REVIEWS MOLECULAR CELL BIOLOGY (2010)
MECHANISMS OF DISEASE Susceptibility Pathways in Fanconi's Anemia and Breast Cancer
Alan D. D'Andrea
NEW ENGLAND JOURNAL OF MEDICINE (2010)
FAN1 Acts with FANCI-FANCD2 to Promote DNA Interstrand Cross-Link Repair
Ting Liu et al.
SCIENCE (2010)
Roles of RECQ helicases in recombination based DNA repair, genomic stability and aging
Dharmendra Kumar Singh et al.
BIOGERONTOLOGY (2009)
Topo III alpha and BLM Act within the Fanconi Anemia Pathway in Response to DNA-Crosslinking Agents
A. W. Hemphill et al.
CYTOGENETIC AND GENOME RESEARCH (2009)
Human RPA phosphorylation by ATR stimulates DNA synthesis and prevents ssDNA accumulation during DNA-replication stress
Vitaly M. Vassin et al.
JOURNAL OF CELL SCIENCE (2009)
Checkpoint Signaling from a Single DNA Interstrand Crosslink
Merav Ben-Yehoyada et al.
MOLECULAR CELL (2009)
FANCM Connects the Genome Instability Disorders Bloom's Syndrome and Fanconi Anemia
Andrew J. Deans et al.
MOLECULAR CELL (2009)
The Fanconi Anemia Pathway Promotes Replication-Dependent DNA Interstrand Cross-Link Repair
Puck Knipscheer et al.
SCIENCE (2009)
More complexity to the Bloom's syndrome complex
Yilun Liu et al.
GENES & DEVELOPMENT (2008)
BLAP18/RMI2, a novel OB-fold-containing protein, is an essential component of the Bloom helicase-double Holliday junction dissolvasome
Thiyam Ramsing Singh et al.
GENES & DEVELOPMENT (2008)
RMI, a new OB-fold complex essential for Bloom syndrome protein to maintain genome stability
Dongyi Xu et al.
GENES & DEVELOPMENT (2008)
Emergence of a DNA-damage response network consisting of Fanconi anaemia and BRCA proteins
Weidong Wang
NATURE REVIEWS GENETICS (2007)
Binding and activation of DNA topoisomerase III by the Rmi1 subunit
Chi-Fu Chen et al.
JOURNAL OF BIOLOGICAL CHEMISTRY (2007)
Endogenous γ-H2AX-ATM-Chk2 checkpoint activation in Bloom's syndrome helicase-deficient cells is related to DNA replication arrested forks
V. Ashutosh Rao et al.
MOLECULAR CANCER RESEARCH (2007)
Role for BLM in replication-fork restart and suppression of origin firing after replicative stress
Sally L. Davies et al.
NATURE STRUCTURAL & MOLECULAR BIOLOGY (2007)
DNA structure-induced recruitment and activation of the Fanconi anemia pathway protein FANCD2
A. Sobeck et al.
MOLECULAR AND CELLULAR BIOLOGY (2007)
Identification of the FANCI protein, a monoubiquitinated FANCD2 paralog required for DNA repair
Agata Smogorzewska et al.
CELL (2007)
Non-denaturing fluorescence in situ hybridization to find replication origins in a specific genome region on the DNA fiber
Kazuto Sugimura et al.
BIOSCIENCE BIOTECHNOLOGY AND BIOCHEMISTRY (2007)
RPA2 is a direct downstream target for ATR to regulate the S-phase checkpoint
Erin Olson et al.
JOURNAL OF BIOLOGICAL CHEMISTRY (2006)
Functions of human replication protein A (RPA): From DNA replication to DNA damage and stress responses
Yue Zou et al.
JOURNAL OF CELLULAR PHYSIOLOGY (2006)
A double Holliday junction dissolvasome comprising BLM, topoisomerase IIIα, and BLAP75
Steven Raynard et al.
JOURNAL OF BIOLOGICAL CHEMISTRY (2006)
BLAP75/RMI1 promotes the BLM-dependent dissolution of homologous recombination intermediates
L Wu et al.
PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA (2006)
Fanconi anemia proteins are required to prevent accumulation of replication-associated DNA double-strand breaks
A Sobeck et al.
MOLECULAR AND CELLULAR BIOLOGY (2006)
A human ortholog of archaeal DNA repair protein Hef is defective in Fanconi anemia complementation group M
AR Meetei et al.
NATURE GENETICS (2005)
The vertebrate Hef ortholog is a component of the Fanconi anemia tumor-suppressor pathway
G Mosedale et al.
NATURE STRUCTURAL & MOLECULAR BIOLOGY (2005)
BLAP75, an essential component of Bloom's syndrome protein complexes that maintain genome integrity
JH Yin et al.
EMBO JOURNAL (2005)
Functional relationships of FANCC to homologous recombination, translesion synthesis, and BLM
S Hirano et al.
EMBO JOURNAL (2005)
Phosphorylation of the Bloom's syndrome helicase and its role in recovery from S-phase arrest
SL Davies et al.
MOLECULAR AND CELLULAR BIOLOGY (2004)
BLM and the FANC proteins collaborate in a common pathway in response to stalled replication forks
P Pichierri et al.
EMBO JOURNAL (2004)
Absence of BLM leads to accumulation of chromosomal DNA breaks during both unperturbed and disrupted S phases
WH Li et al.
JOURNAL OF CELL BIOLOGY (2004)
The Bloom's syndrome helicase suppresses crossing over during homologous recombination
L Wu et al.
NATURE (2003)
A multiprotein nuclear complex connects Fanconi anemia and Bloom syndrome
AR Meetei et al.
MOLECULAR AND CELLULAR BIOLOGY (2003)
S-phase-specific interaction of the Fanconi anemia protein, FANCD2, with BRCA1 and RAD51
T Taniguchi et al.
BLOOD (2002)
Interaction of the fanconi anemia proteins and BRCA1 in a common pathway
I Garcia-Higuera et al.
MOLECULAR CELL (2001)