4.7 Article

Dynamin I phosphorylation by GSK3 controls activity-dependent bulk endocytosis of synaptic vesicles

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NATURE NEUROSCIENCE
卷 13, 期 7, 页码 845-U85

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NATURE PUBLISHING GROUP
DOI: 10.1038/nn.2571

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资金

  1. Wellcome Trust [070569, 084277]
  2. Epilepsy Research UK [0503]
  3. Cancer Institute NSW
  4. Ramaciotti Foundation
  5. Australian National Health and Medical Research Council
  6. Alzheimer's Research Trust [ART/PG2007/4]
  7. MRC [88158]
  8. Danish Agency for Science, Technology and Innovation [274-08-0066]
  9. Australian NHMRC [454886]
  10. Alzheimers Research UK [ART-PG2005-1] Funding Source: researchfish
  11. Biotechnology and Biological Sciences Research Council [C18727] Funding Source: researchfish

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Glycogen synthase kinase 3 (GSK3) is a critical enzyme in neuronal physiology; however, it is not yet known whether it has any specific role in presynaptic function. We found that GSK3 phosphorylates a residue on the large GTPase dynamin I (Ser-774) both in vitro and in primary rat neuronal cultures. This was dependent on prior phosphorylation of Ser-778 by cyclin-dependent kinase 5. Using both acute inhibition with pharmacological antagonists and silencing of expression with short hairpin RNA, we found that GSK3 was specifically required for activity-dependent bulk endocytosis (ADBE) but not clathrin-mediated endocytosis. Moreover we found that the specific phosphorylation of Ser-774 on dynamin I by GSK3 was both necessary and sufficient for ADBE. These results demonstrate a presynaptic role for GSK3 and they indicate that a protein kinase signaling cascade prepares synaptic vesicles for retrieval during elevated neuronal activity.

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