相关参考文献
注意:仅列出部分参考文献,下载原文获取全部文献信息。Th17 and Regulatory T Cells in Mediating and Restraining Inflammation
Dan R. Littman et al.
CELL (2010)
EAE mediated by a non-IFN-γ/non-IL-17 pathway
Mark A. Kroenke et al.
EUROPEAN JOURNAL OF IMMUNOLOGY (2010)
Generation of pathogenic TH17 cells in the absence of TGF-β signalling
Kamran Ghoreschi et al.
NATURE (2010)
Circulating Ly-6C+ myeloid precursors migrate to the CNS and play a pathogenic role during autoimmune demyelinating disease
Irah L. King et al.
BLOOD (2009)
IL-23-driven encephalo-tropism and Th17 polarization during CNS-inflammation in vivo
Gabor Gyuelveszi et al.
EUROPEAN JOURNAL OF IMMUNOLOGY (2009)
IL-27 Blocks RORc Expression to Inhibit Lineage Commitment of Th17 Cells
Caroline Diveu et al.
JOURNAL OF IMMUNOLOGY (2009)
The interleukin 23 receptor is essential for the terminal differentiation of interleukin 17-producing effector T helper cells in vivo
Mandy J. McGeachy et al.
NATURE IMMUNOLOGY (2009)
IL-21 and IL-21R are not required for development of Th17 cells and autoimmunity in vivo
Ivo Sonderegger et al.
EUROPEAN JOURNAL OF IMMUNOLOGY (2008)
GM-CSF mediates autoimmunity by enhancing IL-6-dependent Th17 cell development and survival
Ivo Sonderegger et al.
JOURNAL OF EXPERIMENTAL MEDICINE (2008)
IL-12- and IL-23-modulated T cells induce distinct types of EAE based on histology, CNS chemokine profile, and response to cytokine inhibition
Mark A. Kroenke et al.
JOURNAL OF EXPERIMENTAL MEDICINE (2008)
Rapid ex vivo isolation and long-term culture of human Th17 cells
Hendrik Streeck et al.
JOURNAL OF IMMUNOLOGICAL METHODS (2008)
IL-22 is expressed by TH17 cells in an IL-23-dependent fashion, but not required for the development of autoimmune encephalomyelitis
Katharina Kreymborg et al.
JOURNAL OF IMMUNOLOGY (2007)
TGF-β and IL-6 drive the production of IL-17 and IL-10 by T cells and restrain TH-17 cell-mediated pathology
Mandy J. McGeachy et al.
NATURE IMMUNOLOGY (2007)
Suppressive effect of IL-27 on encephalitogenic Th17 cells and the effector phase of experimental autoimmune encephalomyelitis
Denise C. Fitzgerald et al.
JOURNAL OF IMMUNOLOGY (2007)
IL-21 initiates an alternative pathway to induce proinflammatory TH17 cells
Thomas Korn et al.
NATURE (2007)
APC-derived cytokines and T cell polarization in autoimmune inflammation
Ilona Gutcher et al.
JOURNAL OF CLINICAL INVESTIGATION (2007)
GM-CSF production by autoreactive T cells is required for the activation of microglial cells and the onset of experimental autoimmune encephalomyelitis
Eugene D. Ponomarev et al.
JOURNAL OF IMMUNOLOGY (2007)
The orphan nuclear receptor RORγt directs the differentiation program of proinflammatory IL-17+ T helper cells
Ivaylo I. Ivanov et al.
CELL (2006)
Reciprocal developmental pathways for the generation of pathogenic effector TH17 and regulatory T cells
E Bettelli et al.
NATURE (2006)
TGFβ in the context of an inflammatory cytokine milieu supports de novo differentiation of IL-17-producing T cells
M Veldhoen et al.
IMMUNITY (2006)
Passive induction of experimental allergic encephalomyelitis
Ingunn M. Stromnes et al.
NATURE PROTOCOLS (2006)
IL-23:: changing the verdict on IL-12 function in inflammation and autoimmunity
K Kreymborg et al.
EXPERT OPINION ON THERAPEUTIC TARGETS (2005)
A distinct lineage of CD4 T cells regulates tissue inflammation by producing interleukin 17
H Park et al.
NATURE IMMUNOLOGY (2005)
Interleukin 17-producing CD4+ effector T cells develop via a lineage distinct from the T helper type 1 and 2 lineages
LE Harrington et al.
NATURE IMMUNOLOGY (2005)
Therapeutic efficacy of IL-17 neutralization in murine experimental autoimmune encephalomyelitis
HH Hofstetter et al.
CELLULAR IMMUNOLOGY (2005)
Dendritic cells permit immune invasion of the CNS in an animal model of multiple sclerosis
M Greter et al.
NATURE MEDICINE (2005)
IFN-γ determines distinct clinical outcomes in autoimmune encephalomyelitis
AK Wensky et al.
JOURNAL OF IMMUNOLOGY (2005)
IL-23 drives a pathogenic T cell population that induces autoimmune inflammation
CL Langrish et al.
JOURNAL OF EXPERIMENTAL MEDICINE (2005)
Loss of T-bet, but not STAT1, prevents the development of experimental autoimmune encephalomyelitis
E Bettelli et al.
JOURNAL OF EXPERIMENTAL MEDICINE (2004)
IL-23 produced by CNS-resident cells controls T cell encephalitogenicity during the effector phase of experimental autoimmune encephalomyelitis
B Becher et al.
JOURNAL OF CLINICAL INVESTIGATION (2003)
T-bet is required for optimal production of IFN-γ and antigen-specific T cell activation by dendritic cells
G Lugo-Villarino et al.
PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA (2003)
Interleukin-23 rather than interleukin-12 is the critical cytokine for autoimmune inflammation of the brain
DJ Cua et al.
NATURE (2003)
Interleukin-23 promotes a distinct CD4 T cell activation state characterized by the production of interleukin-17
S Aggarwal et al.
JOURNAL OF BIOLOGICAL CHEMISTRY (2003)
IL-12p35-deficient mice are susceptible to experimental autoimmune encephalomyelitis: Evidence for redundancy in the IL-12 system in the induction of central nervous system autoimmune demyelination
B Gran et al.
JOURNAL OF IMMUNOLOGY (2002)
Experimental autoimmune encephalitis and inflammation in the absence of interleukin-12
B Becher et al.
JOURNAL OF CLINICAL INVESTIGATION (2002)
Granulocyte macrophage colony-stimulating factor: A new putative therapeutic target in multiple sclerosis
JL McQualter et al.
JOURNAL OF EXPERIMENTAL MEDICINE (2001)
Novel p19 protein engages IL-12p40 to form a cytokine, IL-23, with biological activities similar as well as distinct from IL-12
B Oppmann et al.
IMMUNITY (2000)
Failure to suppress the expansion of the activated CD4 T cell population in interferon γ-deficient mice leads to exacerbation of experimental autoimmune encephalomyelitis
CQ Chu et al.
JOURNAL OF EXPERIMENTAL MEDICINE (2000)