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The insulin-specific T cells of nonobese diabetic mice recognize a weak MHC-binding segment in more than one form
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Natural peptides selected by diabetogenic DQ8 and murine I-Ag7molecules show common sequence specificity
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Mechanisms underlying resistance of pancreatic islets from ALR/Lt mice to cytokine-induced destruction
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T-cell receptor transgenic response to an endogenous polymorphic autoantigen determines susceptibility to diabetes
ME Pauza et al.
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Mimotopes for alloreactive and conventional T cells in a peptide-MHC display library
F Crawford et al.
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Cutting edge: The conversion of arginine to citrulline allows for a high-affinity peptide interaction with the rheumatoid arthritis-associated HLA-DRB1*0401 MHC class II molecule
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Evidence for shared recognition of a peptide ligand by a diverse panel of non-obese diabetic mice-derived, islet-specific, diabetogenic T cell clones
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