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注意:仅列出部分参考文献,下载原文获取全部文献信息。Treatment of Neuropathic Pain: An Overview of Recent Guidelines
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Periganglionic inflammation elicits a distally radiating pain hypersensitivity by promoting COX-2 induction in the dorsal root ganglion
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Antibodies to nerve growth factor reverse established tactile allodynia in rodent models of neuropathic pain without tolerance
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Augmented lipopolysaccharide-induced TNF-α production by peritoneal macrophages in type 2 diabetic mice is dependent on elevated glucose and requires p38 MAPK
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Diabetes-induced mechanical hyperalgesia involves spinal mitogen-activated protein kinase activation in neurons and microglia via N-methyl-D-aspartate-dependent mechanisms
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Elevated glucose and diabetes promote interleukin-12 cytokine gene expression in mouse macrophages
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Acute p38-mediated modulation of tetrodotoxin-resistant sodium channels in mouse sensory neurons by tumor necrosis factor-α
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Essential role of Rac1/NADPH oxidase in nerve growth factor induction of TRPV1 expression
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Activation of p38 MAPK in primary afferent neurons by noxious stimulation and its involvement in the development of thermal hyperalgesia
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Activation of p38 mitogen-activated protein kinase in spinal hyperactive microglia contributes to pain hypersensitivity following peripheral nerve injury
M Tsuda et al.
GLIA (2004)
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p38 MAPK activation by NGF in primary sensory neurons after inflammation increases TRPV1 levels and maintains heat hyperalgesia
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Nerve growth factor induces P2X3 expression in sensory neurons
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