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Veronica Costa et al.
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Giselle M. Perry et al.
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Mitochondrial loss, dysfunction and altered dynamics in Huntington's disease
Jinho Kim et al.
HUMAN MOLECULAR GENETICS (2010)
Role of mitochondrial dysfunction in the pathogenesis of Huntington's disease
Rodrigo A. Quintanilla et al.
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Paradoxical delay in the onset of disease caused by super-long CAG repeat expansions in R6/2 mice
A. Jennifer Morton et al.
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Systematic behavioral evaluation of Huntington's disease transgenic and knock-in mouse models
Liliana Menalled et al.
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Impairing the Mitochondrial Fission and Fusion Balance: A New Mechanism of Neurodegeneration
Andrew B. Knott et al.
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Type 2 transglutaminase differentially modulates striatal cell death in the presence of wild type or mutant huntingtin
Qingmin Ruan et al.
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Andrea Rasola et al.
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Michael Forte et al.
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Andrey Y. Abramov et al.
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Mutant huntingtin expression induces mitochondrial calcium handling defects in clonal striatal cells - Functional consequences
Tamara Milakovic et al.
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Mitochondrial-dependent Ca2+ handling in Huntington's disease striatal cells:: Effect of histone deacetylase inhibitors
Jorge M. A. Oliveira et al.
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Transcriptional repression of PGC-α by mutant huntingtin leads to mitochondrial dysfunction and neurodegeneration
Libin Cui et al.
CELL (2006)
Mutant huntingtin aggregates impair mitochondrial movement and trafficking in cortical neurons
DTW Chang et al.
NEUROBIOLOGY OF DISEASE (2006)
The ubiquitin-proteasome system in Huntington's disease
AG Valera et al.
NEUROSCIENTIST (2005)
Mitochondrial respiration and ATP production are significantly impaired in striatal cells expressing mutant Huntingtin
T Milakovic et al.
JOURNAL OF BIOLOGICAL CHEMISTRY (2005)
Oxidative stress is involved in the permeabilization of the inner membrane of brain mitochondria exposed to hypoxia/reoxygenation and low micromolar Ca2+
L Schild et al.
FEBS JOURNAL (2005)
Properties of the permeability transition pore in mitochondria devoid of cyclophilin D
E Basso et al.
JOURNAL OF BIOLOGICAL CHEMISTRY (2005)
Neurotoxic calcium transfer from endoplasmic reticulum to mitochondria is regulated by cyclin-dependent kinase 5-dependent phosphorylation of tau
F Darios et al.
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Striatal cells from mutant huntingtin knock-in mice are selectively vulnerable to mitochondrial complex II inhibitor-induced cell death through a non-apoptotic pathway
QM Ruan et al.
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S Vesce et al.
JOURNAL OF NEUROCHEMISTRY (2004)
Mutant huntingtin directly increases susceptibility of mitochondria to the calcium-induced permeability transition and cytochrome c release
YS Choo et al.
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Calcium, ATP, and ROS: a mitochondrial love-hate triangle
PS Brookes et al.
AMERICAN JOURNAL OF PHYSIOLOGY-CELL PHYSIOLOGY (2004)
Mechanism of neurodegenerative disease: role of the ubiquitin proteasome system
L Petrucelli et al.
ANNALS OF MEDICINE (2004)
Staying in aerobic shape: how the structural integrity of mitochondria and mitochondrial DNA is maintained
SV Scott et al.
CURRENT OPINION IN CELL BIOLOGY (2003)
Huntingtin and Huntingtin-associated protein 1 influence neuronal calcium signaling mediated by inositol-(1,4,5) triphosphate receptor type 1
TS Tang et al.
NEURON (2003)
Mitochondrial dynamics and division in budding yeast
JM Shaw et al.
TRENDS IN CELL BIOLOGY (2002)
Loss of normal huntingtin function: new developments in Huntington's disease research
E Cattaneo et al.
TRENDS IN NEUROSCIENCES (2001)
Dominant phenotypes produced by the HD mutation in STHdhQ111 striatal cells
F Trettel et al.
HUMAN MOLECULAR GENETICS (2000)