4.6 Article

Mitochondrial permeability transition pore induces mitochondria injury in Huntington disease

相关参考文献

注意:仅列出部分参考文献,下载原文获取全部文献信息。
Article Biochemistry & Molecular Biology

Dysregulation of Mitochondrial Calcium Signaling and Superoxide Flashes Cause Mitochondrial Genomic DNA Damage in Huntington Disease

Jiu-Qiang Wang et al.

JOURNAL OF BIOLOGICAL CHEMISTRY (2013)

Article Biochemistry & Molecular Biology

p53 Opens the Mitochondrial Permeability Transition Pore to Trigger Necrosis

Angelina V. Vaseva et al.

Editorial Material Biochemistry & Molecular Biology

Measuring reactive oxygen and nitrogen species with fluorescent probes: challenges and limitations

Balaraman Kalyanaraman et al.

FREE RADICAL BIOLOGY AND MEDICINE (2012)

Article Biochemistry & Molecular Biology

Mitochondrial calcium uptake capacity as a therapeutic target in the R6/2 mouse model of Huntington's disease

Giselle M. Perry et al.

HUMAN MOLECULAR GENETICS (2010)

Article Biochemistry & Molecular Biology

Mitochondrial loss, dysfunction and altered dynamics in Huntington's disease

Jinho Kim et al.

HUMAN MOLECULAR GENETICS (2010)

Review Neurosciences

Role of mitochondrial dysfunction in the pathogenesis of Huntington's disease

Rodrigo A. Quintanilla et al.

BRAIN RESEARCH BULLETIN (2009)

Article Neurosciences

Paradoxical delay in the onset of disease caused by super-long CAG repeat expansions in R6/2 mice

A. Jennifer Morton et al.

NEUROBIOLOGY OF DISEASE (2009)

Article Neurosciences

Systematic behavioral evaluation of Huntington's disease transgenic and knock-in mouse models

Liliana Menalled et al.

NEUROBIOLOGY OF DISEASE (2009)

Article Cell Biology

Impairing the Mitochondrial Fission and Fusion Balance: A New Mechanism of Neurodegeneration

Andrew B. Knott et al.

MITOCHONDRIA AND OXIDATIVE STRESS IN NEURODEGENERATIVE DISORDERS (2008)

Article Biochemistry & Molecular Biology

Type 2 transglutaminase differentially modulates striatal cell death in the presence of wild type or mutant huntingtin

Qingmin Ruan et al.

JOURNAL OF NEUROCHEMISTRY (2007)

Article Multidisciplinary Sciences

Cyclophilin D inactivation protects axons in experimental autoimmune encephalomyelitis, an animal model of multiple sclerosis

Michael Forte et al.

PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA (2007)

Article Biochemistry & Molecular Biology

Mutant huntingtin expression induces mitochondrial calcium handling defects in clonal striatal cells - Functional consequences

Tamara Milakovic et al.

JOURNAL OF BIOLOGICAL CHEMISTRY (2006)

Review Clinical Neurology

The ubiquitin-proteasome system in Huntington's disease

AG Valera et al.

NEUROSCIENTIST (2005)

Article Biochemistry & Molecular Biology

Mitochondrial respiration and ATP production are significantly impaired in striatal cells expressing mutant Huntingtin

T Milakovic et al.

JOURNAL OF BIOLOGICAL CHEMISTRY (2005)

Article Biochemistry & Molecular Biology

Properties of the permeability transition pore in mitochondria devoid of cyclophilin D

E Basso et al.

JOURNAL OF BIOLOGICAL CHEMISTRY (2005)

Review Cell Biology

Calcium, ATP, and ROS: a mitochondrial love-hate triangle

PS Brookes et al.

AMERICAN JOURNAL OF PHYSIOLOGY-CELL PHYSIOLOGY (2004)

Article Medicine, General & Internal

Mechanism of neurodegenerative disease: role of the ubiquitin proteasome system

L Petrucelli et al.

ANNALS OF MEDICINE (2004)

Review Cell Biology

Mitochondrial dynamics and division in budding yeast

JM Shaw et al.

TRENDS IN CELL BIOLOGY (2002)

Review Neurosciences

Loss of normal huntingtin function: new developments in Huntington's disease research

E Cattaneo et al.

TRENDS IN NEUROSCIENCES (2001)

Article Biochemistry & Molecular Biology

Dominant phenotypes produced by the HD mutation in STHdhQ111 striatal cells

F Trettel et al.

HUMAN MOLECULAR GENETICS (2000)