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注意:仅列出部分参考文献,下载原文获取全部文献信息。A Yersinia Effector Protein Promotes Virulence by Preventing Inflammasome Recognition of the Type III Secretion System
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CELL HOST & MICROBE (2010)
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The proteasome pathway destabilizes Yersinia outer protein E and represses its antihost cell activities
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Plague bacteria target immune cells during infection
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The plague virulence protein YopM targets the innate immune response by causing a global depletion of NK cells
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A bacterial type III secretion system inhibits actin polymerization to prevent pore formation in host cell membranes
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LcrV, a substrate for Yersinia enterocolitica type III secretion, is required for toxin targeting into the cytosol of HeLa cells
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