期刊
MOLECULAR CELL
卷 56, 期 4, 页码 469-480出版社
CELL PRESS
DOI: 10.1016/j.molcel.2014.11.001
关键词
-
资金
- U.S. National Institutes of Health
- American Lebanese Syrian Associated Charities
Receptor Interacting Protein Kinase-1 (RIPK1), a key player in inflammation and cell death, assumes opposite functions depending on the cellular context and its posttranslational modifications. Genetic evidence supported by biochemical and cellular biology approaches sheds light on the circumstances in which RIPK1 promotes or inhibits these processes.
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