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Reciprocal co-regulation of EGR2 and MECP2 is disrupted in Rett syndrome and autism
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Integrated epigenomic analyses of neuronal MeCP2 reveal a role for long-range interaction with active genes
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The RET oncogene is a critical component of transcriptional programs associated with retinoic acid-induced differentiation in neuroblastoma
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Inhibitors of differentiation (ID1, ID29 ID3 and ID4) genes are neuronal targets of MeCP2 that are elevated in Rett syndrome
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Loss of silent-chromatin looping and impaired imprinting of DLX5 in Rett syndrome
S Horike et al.
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A previously unidentified MECP2 open reading frame defines a new protein isoform relevant to Rett syndrome
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The major form of MeCP2 has a novel N-terminus generated by alternative splicing
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Mild overexpression of MeCP2 causes a progressive neurological disorder in mice
AL Collins et al.
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Derepression of BDNF transcription involves calcium-dependent phosphorylation of MeCP2
WG Chen et al.
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Elevated methyl-CpG-binding protein 2 expression is acquired during postnatal human brain development and is correlated with alternative polyadenylation
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Mice with truncated MeCP2 recapitulate many Rett syndrome features and display hyperacetylation of histone H3
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Insight into Rett syndrome: MeCP2 levels display tissue- and cell-specific differences and correlate with neuronal maturation
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A mouse Mecp2-null mutation causes neurological symptoms that mimic Rett syndrome
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Deficiency of methyl-CpG binding protein-2 in CNS neurons results in a Rett-like phenotype in mice
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