期刊
MEDIATORS OF INFLAMMATION
卷 2011, 期 -, 页码 -出版社
HINDAWI LTD
DOI: 10.1155/2011/652097
关键词
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资金
- Ministerio de Sanidad y Consumo [SAF 2007/63648, 2007/60896, 2009/8367]
- CAM [S2006/GEN-0247]
- Instituto de Salud Carlos III [FIS PI050451, PS09/01405, CP04/00060]
- European Network [HEALTH F2-2008-200647]
- EUROSALUD [EUS2008-03565]
- Red RECAVA [RD06/0014/0035]
- Fundacion Espanola del Corazon
- Sociedad Espanola de Arteriosclerosis
- Fundacion Mutua Madrilena
- MSD
- Pfizer
Diabetic cardiomyopathy entails the cardiac injury induced by diabetes independently of any vascular disease or hypertension. Some transcription factors have been proposed to control the gene program involved in the setting and development of related processes. Nuclear factor-kappa B is a pleiotropic transcription factor associated to the regulation of many heart diseases. However, the nuclear factor-kappa B role in diabetic cardiomyopathy is under investigation. In this paper, we review the nuclear factor-kappa B pathway and its role in several processes that have been linked to diabetic cardiomyopathy, such as oxidative stress, inflammation, endothelial dysfunction, fibrosis, hypertrophy and apoptosis.
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