期刊
KIDNEY INTERNATIONAL
卷 94, 期 4, 页码 650-652出版社
ELSEVIER SCIENCE INC
DOI: 10.1016/j.kint.2018.05.025
关键词
-
Aberrant regulation of an alternative pathway of the complement system could be a therapeutic target of C3 glomerulopathy, including dense deposit disease. In the current issue, Bekassy and colleagues provide data on enzymatic conversion of C3 by renin in vitro and on the efficacy of a direct renin inhibitor, aliskiren, on systemic and renal complement activation in patients with dense deposit disease. Copyright (C) 2018, International Society of Nephrology. Published by Elsevier Inc. All rights reserved.
作者
我是这篇论文的作者
点击您的名字以认领此论文并将其添加到您的个人资料中。
推荐
暂无数据