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注意:仅列出部分参考文献,下载原文获取全部文献信息。Interaction of the Papillomavirus E8∧E2C Protein with the Cellular CHD6 Protein Contributes to Transcriptional Repression
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Inhibition of transcription and DNA replication by the papillomavirus E8ΛE2C protein is mediated by interaction with corepressor molecules
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The E8∧E2 Gene Product of Human Papillomavirus Type 16 Represses Early Transcription and Replication but Is Dispensable for Viral Plasmid Persistence in Keratinocytes
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JOURNAL OF VIROLOGY (2008)
The E8 repression domain can replace the E2 transactivation domain for growth inhibition of HeLa cells by papillomavirus E2 proteins
Frank Stubenrauch et al.
INTERNATIONAL JOURNAL OF CANCER (2007)
Gene expression profiles reveal an upregulation of E2F and downregulation of interferon targets by HPV18 but no changes between keratinocytes with integrated or episomal viral genomes
Bernd Karstensen et al.
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E2 proteins from high- and low-risk human papillomavirus types differ in their ability to bind p53 and induce apoptotic cell death
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JOURNAL OF VIROLOGY (2006)
Nucleo-cytoplasmic shuttling of high risk human Papillomavirus E2 proteins induces apoptosis
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RNA interference of human papillomavirus type 18 E6 and E7 induces senescence in HeLa cells
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ONCOGENE (2003)
The papillomavirus E8 boolean AND E2C protein represses DNA replication from extrachromosomal origins
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Papillomaviruses and cancer: From basic studies to clinical application
H zur Hausen
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SETDB1: a novel KAP-1-associated histone H3, lysine 9-specific methyltransferase that contributes to HP1-mediated silencing of euchromatic genes by KRAB zinc-finger proteins
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The E8 domain confers a novel long-distance transcriptional repression activity on the E8-over-cap-E2C protein of high-risk human papillomavirus type 31
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JOURNAL OF VIROLOGY (2001)
Repression of human papillomavirus oncogenes in HeLa cervical carcinoma cells causes the orderly reactivation of dormant tumor suppressor pathways
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PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA (2000)
Papillomavirus E2 induces senescence in HPV-positive cells via pRB- and p21CIP-dependent pathways
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JOURNAL OF VIROLOGY (2000)
The E8-E2C protein, a negative regulator of viral transcription and replication, is required for extrachromosomal maintenance of human papillomavirus type 31 in keratinocytes
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JOURNAL OF VIROLOGY (2000)
The human papillomavirus (HPV) 16 E2 protein induces apoptosis in the absence of other HPV proteins and via a p59-dependent pathway
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Molecular determinants for targeting heterochromatin protein 1-mediated gene silencing: Direct chromoshadow domain-KAP-1 corepressor interaction is essential
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