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注意:仅列出部分参考文献,下载原文获取全部文献信息。Postradiation cutaneous angiosarcoma after treatment of breast carcinoma is characterized by MYC amplification in contrast to atypical vascular lesions after radiotherapy and control cases: clinicopathological, immunohistochemical and molecular analysis of 66 cases
T. Mentzel et al.
MODERN PATHOLOGY (2012)
Consistent MYC and FLT4 Gene Amplification in Radiation-Induced Angiosarcoma But Not in Other Radiation-Associated Atypical Vascular Lesions
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MYC High Level Gene Amplification Is a Distinctive Feature of Angiosarcomas after Irradiation or Chronic Lymphedema
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AMERICAN JOURNAL OF PATHOLOGY (2010)
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JOURNAL OF BIOLOGICAL CHEMISTRY (2007)
The Kaposi's sarcoma-associated herpesvirus LANA protein stabilizes and activates c-Myc
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Correlation of amplification and overexpression of the c-myc oncogene in high-grade breast cancer:: FISH, in situ hybridisation and immunohistochemical analyses
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c-Myc interacts with hypoxia to induce angiogenesis in vivo by a vascular endothelial growth factor-dependent mechanism
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CANCER RESEARCH (2004)
B lymphocyte-specific c-myc expression stimulates early and functional expansion of the vasculature and lymphatics during lymphomagenesis
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AMERICAN JOURNAL OF PATHOLOGY (2003)
C-myc oncoprotein expression and gene amplification in apocrine metaplasia and apocrine change within sclerosing adenosis of the breast
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c-Myc is essential for vasculogenesis and angiogenesis during development and tumor progression
TA Baudino et al.
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Angiogenesis is an early event in the generation of myc-induced lymphomas
KA Brandvold et al.
ONCOGENE (2000)