期刊
JOURNAL OF THE AMERICAN SOCIETY OF NEPHROLOGY
卷 21, 期 8, 页码 1247-1253出版社
AMER SOC NEPHROLOGY
DOI: 10.1681/ASN.2010060616
关键词
-
资金
- NIDDK NIH HHS [DK73299, DK84077, DK87389, R01DK081576, R03DK081687, K08DK074558] Funding Source: Medline
Epithelial-mesenchymal transition (EMT) is a mechanism for generating primitive mesenchymal cells during gastrulation or mobile tumor cells during cancer metastasis. For 15 years, EMT has also been viewed as a principal source of fibroblasts in tissue fibrosis. Because several recent studies question its role in fibrogenesis, it seems like a good time for debate.
作者
我是这篇论文的作者
点击您的名字以认领此论文并将其添加到您的个人资料中。
推荐
暂无数据