相关参考文献
注意:仅列出部分参考文献,下载原文获取全部文献信息。Activation of proteolytic enzymes and depression of the sarcolemmal Na+/K+-ATPase in ischemia-reperfused heart may be mediated through oxidative stress
Raja B. Singh et al.
CANADIAN JOURNAL OF PHYSIOLOGY AND PHARMACOLOGY (2012)
Pathways of Ca2+ entry and cytoskeletal damage following eccentric contractions in mouse skeletal muscle
Bao-Ting Zhang et al.
JOURNAL OF APPLIED PHYSIOLOGY (2012)
Microtubules Underlie Dysfunction in Duchenne Muscular Dystrophy
Ramzi J. Khairallah et al.
SCIENCE SIGNALING (2012)
Disrupted Junctional Membrane Complexes and Hyperactive Ryanodine Receptors After Acute Junctophilin Knockdown in Mice
Ralph J. van Oort et al.
CIRCULATION (2011)
Activation of skeletal muscle calpain-3 by eccentric exercise in humans does not result in its translocation to the nucleus or cytosol
Robyn M. Murphy et al.
JOURNAL OF APPLIED PHYSIOLOGY (2011)
Junctophilin 1 and 2 Proteins Interact with the L-type Ca2+ Channel Dihydropyridine Receptors (DHPRs) in Skeletal Muscle
Lucia Golini et al.
JOURNAL OF BIOLOGICAL CHEMISTRY (2011)
Junctophilin damage contributes to early strength deficits and EC coupling failure after eccentric contractions
B. T. Corona et al.
AMERICAN JOURNAL OF PHYSIOLOGY-CELL PHYSIOLOGY (2010)
Ischemia-reperfusion-induced changes in sarcolemmal Na+/K+-ATPase are due to the activation of calpain in the heart
Raja B. Singh et al.
CANADIAN JOURNAL OF PHYSIOLOGY AND PHARMACOLOGY (2010)
Skeletal Muscle NADPH Oxidase Is Increased and Triggers Stretch-Induced Damage in the mdx Mouse
Nicholas P. Whitehead et al.
PLOS ONE (2010)
Involvement of calpains in Ca2+-induced disruption of excitation-contraction coupling in mammalian skeletal muscle fibers
Esther Verburg et al.
AMERICAN JOURNAL OF PHYSIOLOGY-CELL PHYSIOLOGY (2009)
Hypernitrosylated ryanodine receptor calcium release channels are leaky in dystrophic muscle
Andrew M. Bellinger et al.
NATURE MEDICINE (2009)
Role of the calcium-calpain pathway in cytoskeletal damage after eccentric contractions
Bao-Ting Zhang et al.
JOURNAL OF APPLIED PHYSIOLOGY (2008)
Conformation-dependent stability of junctophilin 1 (JP1) and ryanodine receptor type 1 (RyRl) channel complex is mediated by their hyper-reactive thiols
Andrew J. Phimister et al.
JOURNAL OF BIOLOGICAL CHEMISTRY (2007)
Ca2+ activation of diffusible and bound pools of μ-calpain in rat skeletal muscle
Robyn M. Murphy et al.
JOURNAL OF PHYSIOLOGY-LONDON (2006)
Uncoupling store-operated Ca2+ entry and altered Ca2+ release from sarcoplasmic reticulum through silencing of junctophilin genes
Yutaka Hirata et al.
BIOPHYSICAL JOURNAL (2006)
Long-lasting muscle fatigue:: partial disruption of excitation-contraction coupling by elevated cytosolic Ca2+ concentration during contractions
E Verburg et al.
AMERICAN JOURNAL OF PHYSIOLOGY-CELL PHYSIOLOGY (2006)
mu-Calpain and calpain-3 are not autolyzed with exhaustive exercise in humans
RM Murphy et al.
AMERICAN JOURNAL OF PHYSIOLOGY-CELL PHYSIOLOGY (2006)
Effects of stretch-activated channel blockers on [Ca2+]i and muscle damage in the mdx mouse
EW Yeung et al.
JOURNAL OF PHYSIOLOGY-LONDON (2005)
Possible regulation of the conventional calpain system by skeletal muscle-specific calpain, p94/calpain 3
Y Ono et al.
JOURNAL OF BIOLOGICAL CHEMISTRY (2004)
Skeletal muscle function: Role of ionic changes in fatigue, damage and disease
DG Allen
CLINICAL AND EXPERIMENTAL PHARMACOLOGY AND PHYSIOLOGY (2004)
The action potential-evoked sarcoplasmic reticulum calcium release is impaired in mdx mouse muscle fibres
CE Woods et al.
JOURNAL OF PHYSIOLOGY-LONDON (2004)
Deficiency of triad formation in developing skeletal muscle cells lacking junctophilin type 1
S Komazaki et al.
FEBS LETTERS (2002)
Deficiency of triad junction and contraction in mutant skeletal muscle lacking junctophilin type 1
K Ito et al.
JOURNAL OF CELL BIOLOGY (2001)