相关参考文献
注意：仅列出部分参考文献，下载原文获取全部文献信息。Liver X receptor agonist treatment regulates inflammatory response after spinal cord trauma
Irene Paterniti et al.
JOURNAL OF NEUROCHEMISTRY (2010)
Liver X Receptor Agonist Treatment Ameliorates Amyloid Pathology and Memory Deficits Caused by High-Fat Diet in APP23 Mice
Nicholas F. Fitz et al.
JOURNAL OF NEUROSCIENCE (2010)
Temporal-spatial expression of presenilin 1 and the production of amyloid-β after acute spinal cord injury in adult rat
S. Kobayashi et al.
NEUROCHEMISTRY INTERNATIONAL (2010)
Simvastatin Therapy Prevents Brain Trauma-Induced Increases in β-Amyloid Peptide Levels
Eric E. Abrahamson et al.
ANNALS OF NEUROLOGY (2009)
Long-term Neurologic Outcomes After Traumatic Brain Injury
Jeffrey J. Bazarian et al.
JOURNAL OF HEAD TRAUMA REHABILITATION (2009)
Amyloid precursor protein secretases as therapeutic targets for traumatic brain injury
David J. Loane et al.
NATURE MEDICINE (2009)
Activation of liver X receptors promotes neuroprotection and reduces brain inflammation in experimental stroke
Jesus R. Morales et al.
CIRCULATION (2008)
Treatment with LXR agonists after focal cerebral ischemia prevents brain damage
Luigi Sironi et al.
FEBS LETTERS (2008)
Increased expression of the γ-secretase components presenilin-1 and nicastrin in activated astrocytes and microglia following traumatic brain injury
Yasmine Nadler et al.
GLIA (2008)
ApoE promotes the proteolytic degradation of Aβ
Qingguang Jiang et al.
NEURON (2008)
Liver X receptor activation enhances cholesterol loss from the brain, decreases neuroinflammation, and increases survival of the NPC1 mouse
Joyce J. Repa et al.
JOURNAL OF NEUROSCIENCE (2007)
The metalloprotease inhibitor TIMP-3 regulates amyloid precursor protein and apolipoprotein E receptor proteolysis
Hyang-Sook Hoe et al.
JOURNAL OF NEUROSCIENCE (2007)
Regulation of central nervous system cholesterol homeostasis by the liver X receptor agonist TO-901317
Gunter P. Eckert et al.
NEUROSCIENCE LETTERS (2007)
Attenuation of neuroinflammation and Alzheimer's disease pathology by liver x receptors
Noam Zelcer et al.
PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA (2007)
Hypoxia-inducible factor 1α (HIF-1α)-mediated hypoxia increases BACE1 expression and β-amyloid generation
Xian Zhang et al.
JOURNAL OF BIOLOGICAL CHEMISTRY (2007)
An apolipoprotein e-based therapeutic improves outcome and reduces Alzheimer's disease pathology following closed head injury: Evidence of pharmacogenomic interaction
H. Wang et al.
NEUROSCIENCE (2007)
The effects of ABCA1 on cholesterol efflux and Aβ levels in vitro and in vivo
Mark P. Burns et al.
JOURNAL OF NEUROCHEMISTRY (2006)
Caspase inhibition therapy abolishes brain trauma-induced increases in Aβ peptide:: Implications for clinical outcome
EE Abrahamson et al.
EXPERIMENTAL NEUROLOGY (2006)
The absence of ABCA1 decreases soluble ApoE levels but does not diminish amyloid deposition in two murine models of Alzheimer disease
V Hirsch-Reinshagen et al.
JOURNAL OF BIOLOGICAL CHEMISTRY (2005)
Lack of ABCA1 considerably decreases brain ApoE level and increases amyloid deposition in APP23 mice
R Koldamova et al.
JOURNAL OF BIOLOGICAL CHEMISTRY (2005)
Deletion of Abca1 increases Aβ deposition in the PDAPP transgenic mouse model of Alzheimer disease
SE Wahrle et al.
JOURNAL OF BIOLOGICAL CHEMISTRY (2005)
The liver X receptor ligand T0901317 decreases amyloid β production in vitro and in a mouse model of Alzheimer's disease
T Koldamova et al.
JOURNAL OF BIOLOGICAL CHEMISTRY (2005)
Experimental traumatic brain injury in rats stimulates the expression, production and activity of Alzheimer's disease β-secretase (BACE-1)
I Blasko et al.
JOURNAL OF NEURAL TRANSMISSION (2004)
Long-term accumulation of amyloid-β, β-secretase, presenilin-1, and caspase-3 in damaged axons following brain trauma
XH Chen et al.
AMERICAN JOURNAL OF PATHOLOGY (2004)
Alzheimer's pathology in human temporal cortex surgically excised after severe brain injury
MD Ikonomovic et al.
EXPERIMENTAL NEUROLOGY (2004)
Expression of liver X receptor target genes decreases cellular amyloid β peptide secretion
Y Sun et al.
JOURNAL OF BIOLOGICAL CHEMISTRY (2003)
Reciprocal regulation of inflammation and lipid metabolism by liver X receptors
SB Joseph et al.
NATURE MEDICINE (2003)
Long-term accumulation of amyloid-β in axons following brain trauma without persistent upregulation of amyloid precursor protein genes
A Iwata et al.
JOURNAL OF NEUROPATHOLOGY AND EXPERIMENTAL NEUROLOGY (2002)
Changes in expression of amyloid precursor protein and interleukin-1β after experimental traumatic brain injury in rats
JR Ciallella et al.
JOURNAL OF NEUROTRAUMA (2002)
Caspase-3-mediated cleavage of amyloid precursor protein and formation of amyloid β peptide in traumatic axonal injury
JR Stone et al.
JOURNAL OF NEUROTRAUMA (2002)
Repetitive mild brain trauma accelerates Aβ deposition, lipid peroxidation, and cognitive impairment in a transgenic mouse model of Alzheimer amyloidosis
K Uryu et al.
JOURNAL OF NEUROSCIENCE (2002)
β-amyloid peptides are cytotoxic to astrocytes in culture:: A role for oxidative stress
B Brera et al.
NEUROBIOLOGY OF DISEASE (2000)
Brain trauma in aged transgenic mice induces regression of established Aβ deposits
Y Nakagawa et al.
EXPERIMENTAL NEUROLOGY (2000)
分享论文
