4.6 Review

Thematic Review Series: Genetics of Human Lipid Diseases Genetic determinants of plasma triglycerides

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Carbohydrate response element binding protein directly promotes lipogenic enzyme gene transcription

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Angiopoietin-like protein 3 mediates hypertriglyceridemia induced by the liver X receptor

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Relative contribution of variation within the APOC3/A4/A5 gene cluster in determining plasma triglycerides

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Two independent apolipoprotein A5 haplotypes influence human plasma triglyceride levels

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ANGPTL3 decreases very low density lipoprotein triglyceride clearance by inhibition of lipoprotein lipase

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Adenoviral overexpression of apolipoprotein A-V reduces serum levels of triglycerides and cholesterol in mice

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Evidence that triglycerides are an independent coronary heart disease risk factor

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