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Classical swine fever virus Npro interacts with interferon regulatory factor 3 and induces its proteasomal degradation
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The NPro product of bovine viral diarrhea virus inhibits DNA binding by interferon regulatory factor 3 and targets it for proteasomal degradation
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The amino-terminal domain of bovine viral diarrhea virus N-pro protein is necessary for alpha/beta interferon antagonism
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Npro of classical swine fever virus is an antagonist of double-stranded RNA-mediated apoptosis and IFN-α/β induction
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Modulation of p53 activity by IκBα:: Evidence suggesting a common phylogeny between NF-κB and p53 transcription factors
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Establishment of a subgenomic replicon for bovine viral diarrhea virus in Huh-7 cells and modulation of interferon-regulated factor 3-mediated antiviral response
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Herpes virus proteins BICP0 and BICP0 can activate NF-κB by catalyzing IκBα ubiquitination
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The vaccinia virus K1L gene product inhibits host NF-κB activation by preventing IκBα degradation
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Classical swine fever virus induces proinflammatory cytokines and tissue factor expression and inhibits apoptosis and interferon synthesis during the establishment of long-term infection of porcine vascular endothelial cells
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JOURNAL OF GENERAL VIROLOGY (2004)
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Recruitment of IκBα to the hes1 promoter is associated with transcriptional repression
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Classical swine fever virus interferes with cellular antiviral defense:: Evidence for a novel function of Npro
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Inhibition of beta interferon transcription by noncytopathogenic bovine viral diarrhea virus is through an interferon regulatory factor 3-dependent mechanism
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The non-ankyrin C terminus of IκBα physically interacts with p53 in vivo and dissociates in response to apoptotic stress, hypoxia, DNA damage, and transforming growth factor-β1-mediated growth suppression
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Establishment of persistent infection with non-cytopathic bovine viral diarrhoea virus in cattle is associated with a failure to induce type 1 interferon
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Noncytopathic bovine viral diarrhea virus inhibits double-stranded RNA-induced apoptosis and interferon synthesis
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Pathogenesis of granulocytopenia and bone marrow atrophy during classical swine fever involves apoptosis and necrosis of uninfected cells
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