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Loss of integrin αvβ8 on dendritic cells causes autoimmunity and colitis in mice
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CD4+ CD5+ regulatory T cells render naive CD4+ CD25-T cells anergic and suppressive
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TGF-β-mediated suppression by CD4+CD25+ T cells is facilitated by CTLA-4 signaling
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The lifestyle of naturally occurring CD4+CD25+Foxp3+ regulatory T cells
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TGF-β1 production by CD4+CD25+ regulatory T cells is not essential for suppression of intestinal inflammation
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T cells that cannot respond to TGF-β escape control by CD4+CD25+ regulatory T cells
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TGF-β1 plays an important role in the mechanism of CD4+CD25+ regulatory T cell activity in both humans and mice
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Induction of foxP3+ regulatory T cells in the periphery of T cell receptor transgenic mice tolerized to transplants
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Conversion of peripheral CD4+CD25- naive T cells to CD4+CD25+ regulatory T cells by TGF-β induction of transcription factor Foxp3
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Foxp3 programs the development and function of CD4+CD25+ regulatory T cells
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Control of regulatory T cell development by the transcription factor Foxp3
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CD4+CD25+ regulatory T cells can mediate suppressor function in the absence of transforming growth factor β1 production and responsiveness
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Human CD4+CD25+ regulatory, contact-dependent T cells induce interleukin 1-producing, contact-independent type 1-like regulatory T cells
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Cell contact-dependent immunosuppression by CD4+CD25+ regulatory T cells is mediated by cell surface-bound transforming growth factor β
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Disruption of a new forkhead/winged-helix protein, scurfin, results in the fatal lymphoproliferative disorder of the scurfy mouse
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The immune dysregulation, polyendocrinopathy, enteropathy, X-linked syndrome (IPEX) is caused by mutations of FOXP3
CL Bennett et al.
NATURE GENETICS (2001)
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