期刊
JOURNAL OF CLINICAL MICROBIOLOGY
卷 49, 期 7, 页码 2680-2684出版社
AMER SOC MICROBIOLOGY
DOI: 10.1128/JCM.02144-10
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- Ministry of Education, Science, Sports, Culture and Technology of Japan [18590438]
- Grants-in-Aid for Scientific Research [18590438] Funding Source: KAKEN
Of 38 vancomycin-intermediate Staphylococcus aureus (VISA) clinical strains, 27 (71%) possessed a mutation(s) in rpoB encoding the beta-subunit of RNA polymerase. Furthermore, 95.6% of the rifampin-resistant mutants obtained from 9 methicillin-resistant S. aureus (MRSA) clinical isolates showed decreased vancomycin susceptibilities. These data indicate the involvement of an rpoB mutation in VISA phenotype expression.
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