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Streamson Chua
ENDOCRINOLOGY (2010)
Large, rare chromosomal deletions associated with severe early-onset obesity
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Obesity and leptin resistance: distinguishing cause from effect
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SH2B1 Enhances Insulin Sensitivity by Both Stimulating the Insulin Receptor and Inhibiting Tyrosine Dephosphorylation of Insulin Receptor Substrate Proteins
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Nucleocytoplasmic Shuttling of the Adapter Protein SH2B1β (SH2-Bβ) Is Required for Nerve Growth Factor (NGF)-Dependent Neurite Outgrowth and Enhancement of Expression of a Subset of NGF-Responsive Genes
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Molecular and neural mediators of leptin action
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Neuronal SH2B1 is essential for controlling energy and glucose homeostasis
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JOURNAL OF CLINICAL INVESTIGATION (2007)
SH2B1 (SH2-B) and JAK2: a multifunctional adaptor protein and kinase made for each other
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TRENDS IN ENDOCRINOLOGY AND METABOLISM (2007)
Hyperphagia, severe obesity, impaired cognitive function, and hyperactivity associated with functional loss of one copy of the brain-derived neurotrophic factor (BDNF) gene
Juliette Gray et al.
DIABETES (2006)
Interaction of SH2-Bβ with RET is involved in signaling of GDNF-induced neurite outgrowth
Y Zhang et al.
JOURNAL OF CELL SCIENCE (2006)
Identification of SH2-B as a key regulator of leptin sensitivity, energy balance, and body weight in mice
DC Ren et al.
CELL METABOLISM (2005)
Tyrosine 813 is a site of JAK2 autophosphorylation critical for activation of JAK2 by SH2-Bβ
JH Kurzer et al.
MOLECULAR AND CELLULAR BIOLOGY (2004)
Clinical spectrum of obesity and mutations in the melanocortin 4 receptor gene
IS Farooqi et al.
NEW ENGLAND JOURNAL OF MEDICINE (2003)
SH2-Bβ is a Rac-binding protein that regulates cell motility
M Diakonova et al.
JOURNAL OF BIOLOGICAL CHEMISTRY (2002)
Four PSM/SH2-B alternative splice variants and their differential roles in mitogenesis
N Yousaf et al.
JOURNAL OF BIOLOGICAL CHEMISTRY (2001)
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