期刊
JOURNAL OF CELL BIOLOGY
卷 203, 期 2, 页码 175-186出版社
ROCKEFELLER UNIV PRESS
DOI: 10.1083/jcb.201306030
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资金
- Programa de Desarrollo de las Ciencias Basicas (PEDECIBA)
- Comision Sectorial de Investigacion Cientifica (CSIC)-UdelaR
- RIKEN Brain Science Institute
- European Union
- Japan Society for the Promotion of Science
Synaptic plasticity, a change in the efficacy of synaptic signaling, is a key property of synaptic communication that is vital to many brain functions. Hebbian forms of long-lasting synaptic plasticity-long-term potentiation (LTP) and long-term depression (LTD)-have been well studied and are considered to be the cellular basis for particular types of memory. Recently, homeostatic synaptic plasticity, a compensatory form of synaptic strength change, has attracted attention as a cellular mechanism that counteracts changes brought about by LTP and LTD to help stabilize neuronal network activity. New findings on the cellular mechanisms and molecular players of the two forms of plasticity are uncovering the interplay between them in individual neurons.
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