期刊
JOURNAL OF BIOLOGICAL CHEMISTRY
卷 288, 期 37, 页码 26505-26511出版社
AMER SOC BIOCHEMISTRY MOLECULAR BIOLOGY INC
DOI: 10.1074/jbc.R113.457564
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资金
- Wellcome Trust [083643/Z/07/Z]
- European Research Council [281348]
- EMBO Young Investigators Programme
- Medical Research Council (MRC) Centre for Obesity and Related Metabolic Disorders
- National Institute for Health Research (NIHR) Cambridge Biomedical Research Centre
- Medical Research Council [G0600717, G0600717B, MC_UU_12012/5/B] Funding Source: researchfish
- MRC [G0600717] Funding Source: UKRI
- Wellcome Trust [083643/Z/07/Z] Funding Source: Wellcome Trust
- European Research Council (ERC) [281348] Funding Source: European Research Council (ERC)
Living organisms possess biological clocks that resonate with environmental cycles in light, temperature, and food availability. Recently, circadian oscillations in the redox state of peroxiredoxin have been described as an additional non-transcriptional timekeeping mechanism. Of note, this redox cycle is conserved in both prokaryotes and eukaryotes. How the classical transcription-translation feedback loop model and this redox oscillation are related is still poorly understood. In this minireview, we describe the most recent evidence pointing to cross-talk between the circadian clock and the redox status of the cell.
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