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Protein studies in dysferlinopathy patients using llama-derived antibody fragments selected by phage display
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Mutant MyoD lacking Cdc2 phosphorylation sites delays M-phase entry
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Null mutation of calpain 3 (p94) in mice causes abnormal sarcomere formation in vivo and in vitro
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Calpain 3 is activated through autolysis within the active site and lyses sarcomeric and sarcolemmal components
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