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Parkin is recruited selectively to impaired mitochondria and promotes their autophagy
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Pink1 regulates mitochondrial dynamics through interaction with the fission/fusion machinery
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Loss-of-function of human PINK1 results in mitochondrial pathology and can be rescued by parkin
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Phosphorylation and cleavage of presenilin-associated rhomboid-like protein (PARL) promotes changes in mitochondrial morphology
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Improved monomeric red, orange and yellow fluorescent proteins derived from Discosoma sp red fluorescent protein
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Alterations in the common fragile site gene Parkin in ovarian and other cancers
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Parkin suppresses dopaminergic neuron-selective neurotoxicity induced by Pael-R in Drosophila
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