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注意：仅列出部分参考文献，下载原文获取全部文献信息。POT1 stimulates RecQ helicases WRN and BLM to unwind telomeric DNA substrates
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The Bloom syndrome helicase BLM interacts with TRF2 in ALT cells and promotes telomeric DNA synthesis
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Bloom's syndrome protein is required for correct relocalization of RAD50/MRE11/NBS1 complex after replication fork arrest
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The N-terminal region of Sgs1, which interacts with Top3, is required for complementation of MMS sensitivity and suppression of hyper-recombination in sgs1 disruptants
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The Bloom's and Werner's syndrome proteins are DNA structure-specific helicases
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Stable association of hsp90 and p23, but not hsp70, with active human telomerase
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SGS1, the Saccharomyces cerevisiae homologue of BLM and WRN, suppresses genome instability and homeologous recombination
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Telomere maintenance by recombination in human cells
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Telomerase-associated protein TEP1 is not essential for telomerase activity or telomere length maintenance in vivo
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NBS1 and TRF1 colocalize at promyelocytic leukemia bodies during late S/G2 phases in immortalized telomerase-negative cells -: Implication of NBS1 in alternative lengthening of telomeres
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Cell-cycle-regulated association of RAD50/MRE11/NBS1 with TRF2 and human telomeres
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Cell cycle regulation of the endogenous wild type Bloom's syndrome DNA helicase
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The Bloom's syndrome gene product interacts with topoisomerase III
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