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注意:仅列出部分参考文献,下载原文获取全部文献信息。Specific requirement for Bax, not Bak, in Myc-induced apoptosis and tumor suppression in vivo
TB Dansen et al.
JOURNAL OF BIOLOGICAL CHEMISTRY (2006)
Cleavage of Bax to p18 Bax accelerates stress-induced apoptosis, and a cathepsin-like protease may rapidly degrade p18 Bax
XF Cao et al.
BLOOD (2003)
被撤回的出版物: Cytoprotective membrane-permeable peptides designed from the Bax-binding domain of Ku70 (Retracted article. See vol 9, pg 480, 2007)
M Sawada et al.
NATURE CELL BIOLOGY (2003)
14-3-3 interacts directly with and negatively regulates pro-apoptotic bax
M Nomura et al.
JOURNAL OF BIOLOGICAL CHEMISTRY (2003)
An integrated database of genes responsive to the Myc oncogenic transcription factor: identification of direct genomic targets
KI Zeller et al.
GENOME BIOLOGY (2003)
A novel Apaf-1-independent putative caspase-2 activation complex
SH Read et al.
JOURNAL OF CELL BIOLOGY (2002)
c-Myc functionally cooperates with Bax to induce apoptosis
P Juin et al.
MOLECULAR AND CELLULAR BIOLOGY (2002)
Requirement for caspase-2 in stress-induced apoptosis before mitochondrial permeabilization
P Lassus et al.
SCIENCE (2002)
Caspase-2 acts upstream of mitochondria to promote cytochrome c release during etoposide-induced apoptosis
JD Robertson et al.
JOURNAL OF BIOLOGICAL CHEMISTRY (2002)
Apoptosis-associated release of Smac/DIABLO from mitochondria requires active caspases and is blocked by Bcl-2
C Adrain et al.
EMBO JOURNAL (2001)
Myc potentiates apoptosis by stimulating Bax activity at the mitochondria
EL Soucie et al.
MOLECULAR AND CELLULAR BIOLOGY (2001)
Proapoptotic BAX and BAK: A requisite gateway to mitochondrial dysfunction and death
MC Wei et al.
SCIENCE (2001)
Cytochrome c maintains mitochondrial transmembrane potential and ATP generation after outer mitochondrial membrane permeabilization during the apoptotic process
NJ Waterhouse et al.
JOURNAL OF CELL BIOLOGY (2001)
Bax is present as a high molecular weight oligomer/complex in the mitochondrial membrane of apoptotic cells
B Antonsson et al.
JOURNAL OF BIOLOGICAL CHEMISTRY (2001)
DIABLO promotes apoptosis by removing MIHA/XIAP from processed caspase 9
PG Ekert et al.
JOURNAL OF CELL BIOLOGY (2001)
Structure of Bax: Coregulation of dimer formation and intracellular localization
M Suzuki et al.
CELL (2000)
Bax oligomerization is required for channel-forming activity in liposomes and to trigger cytochrome c release from mitochondria
B Antonsson et al.
BIOCHEMICAL JOURNAL (2000)